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World J Stem Cells. Sep 26, 2025; 17(9): 109715
Published online Sep 26, 2025. doi: 10.4252/wjsc.v17.i9.109715
Exploring the critical therapeutic window: Dose-frequency optimization of human umbilical cord mesenchymal stem cells for preclinical asthma treatment
Qiong-Hua Chen, Jing-Yang Zheng, Yu-Qin Zhu, Jun-Yao Zhang, Chun-Yan Lin, Xue-E Zhuang, Jing Cheng, Xiao-Yi Huang
Qiong-Hua Chen, Jing-Yang Zheng, The Graduate School of Fujian Medical University, Fuzhou 350122, Fujian Province, China
Qiong-Hua Chen, Jing-Yang Zheng, Jun-Yao Zhang, Chun-Yan Lin, Xue-E Zhuang, Department of Respiratory Medicine, Quanzhou Maternal and Child Health Hospital (Quanzhou Children’s Hospital), Quanzhou 362000, Fujian Province, China
Yu-Qin Zhu, Department of Pathology, Quanzhou Maternal and Child Health Hospital (Quanzhou Children’s Hospital), Quanzhou 362000, Fujian Province, China
Jing Cheng, Animal Experimental Centre, Quanzhou Medical College, Quanzhou 362000, Fujian Province, China
Xiao-Yi Huang, Department of Pharmacology, Quanzhou Medical College, Quanzhou 362000, Fujian Province, China
Author contributions: Chen QH contributed to the funding acquisition, writing-review & editing and supervision of this manuscript; Zheng JY revised and supervised the manuscript; Chen QH, Zhu YQ, Zhang JY, Cheng J, and Huang XY carried out the experiments; Chen QH, Zheng JY, Zhu YQ, Zhang YJ, Lin CY, and Zhuang XE analyzed the data.
Supported by the Joint Innovation Project Funds of Huaqiao University, No. 2022YX001.
Institutional animal care and use committee statement: All procedures involving animals were reviewed and approved by the Quanzhou Children’s Hospital Institutional Review Board [Approval No. 2022(41)].
Conflict-of-interest statement: All the authors report no relevant conflicts of interest for this article.
ARRIVE guidelines statement: The authors have read the ARRIVE guidelines, and the manuscript was prepared and revised according to the ARRIVE guidelines.
Data sharing statement: Technical appendix, statistical code, and dataset available from the corresponding author at cqionghua283@163.com.
Open Access: This article is an open-access article that was selected by an in-house editor and fully peer-reviewed by external reviewers. It is distributed in accordance with the Creative Commons Attribution NonCommercial (CC BY-NC 4.0) license, which permits others to distribute, remix, adapt, build upon this work non-commercially, and license their derivative works on different terms, provided the original work is properly cited and the use is non-commercial. See: https://creativecommons.org/Licenses/by-nc/4.0/
Corresponding author: Qiong-Hua Chen, The Graduate School of Fujian Medical University, No. 1 Xuefu North Road, Shangjie Town, Minhou County, Fuzhou 350122, Fujian Province, China. cqionghua283@163.com
Received: May 19, 2025
Revised: June 17, 2025
Accepted: August 29, 2025
Published online: September 26, 2025
Processing time: 128 Days and 2.8 Hours
Abstract
BACKGROUND

Current drugs primarily target inflammation control but do not reverse tissue remodeling changes for asthma. Human mesenchymal stem cells are known for their anti-inflammatory and tissue remodeling capabilities. However, limited research has explored the therapeutic impact of varying doses and frequencies of human umbilical cord blood-derived mesenchymal stem cells (HUC-MSCs) on established airway remodeling in experimental asthma.

AIM

To explore and optimize the dosage and administration frequency of HUC-MSCs in experimental models of ovalbumin (OVA)-induced asthma.

METHODS

BALB/c mice underwent sensitization and were challenged using OVA. Control animals were administered a saline solution following the same protocol. HUC-MSCs were identified using flow cytometry. HUC-MSCs at incremental dosages (1 × 105, 2 × 105, 4 × 105) were injected via tail veins on day 30 (the second after the final stimulation). After comparing each group and determining the optimal dose, supplement the optimal dose twice on day 30 and day 33 (the second and fifth day after the final stimulation). Bronchoalveolar lavage fluid (BALF) and serum were harvested for analysis of concentrations of interleukin-4 (IL-4), IL-13, immunoglobulin E and interferon-gamma (IFN-γ) by enzyme-linked immunosorbent assay. Pharmacology of airways and lung functions were also evaluated to identify the optimal group.

RESULTS

The study shows that HUC-MSC transplantation ameliorates OVA-induced asthma by significantly reducing airway inflammation and obstruction in preclinical models. This effect is associated with decreased Th2 cytokines IL-4 and IL-13, and increased Th1 cytokine IFN-γ. The optimal dose of 2 × 105 cells/mouse was identified as the most effective in reducing local asthmatic airway inflammation and changing levels of IL-4, IL-13, and IFN-γ in serum and BALF compared to other single doses of HUC-MSC. Multiple treatments with the medium dose (2 × 105 cells) of HUC-MSCs on days 30 and 33 yield the best pathological and lung function outcomes. However, double treatments do not reduce IL-4 and IL-13 expression or enhance IFN-γ production in serum or BALF more effectively than a single medium dose.

CONCLUSION

HUC-MSCs effectively regulate pro-inflammatory mediators in serum and BALF, modulating airway remodeling and lung function. In this acute mouse asthma model, a single dosage of 2 × 105 is optimal, with more significant effects of decreasing airway obstruction requiring repeated administration.

Keywords: Asthma; Human umbilical cord mesenchymal stem cells; Mouse; Dose; Frequency

Core Tip: This study reports, human umbilical cord blood-derived mesenchymal stem cell (HUC-MSC) therapy shows potential for treating asthma with high purity. HUC-MSC has been demonstrated to modulate airway remodeling, enhance lung function, and inhibit the inflammatory response in acute ovalbumin-induced asthma model. This study provides appropriate reference dosages to explore the possible intervention of HUC-MSC further. A single dosage of 2 × 105 cells is optimal, while double treatments with 2 × 105 cells may be optimal in reducing airway obstruction, but not for modulating airway inflammation.