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Case Report
Copyright ©The Author(s) 2026.
World J Clin Cases. Jan 16, 2026; 14(2): 116519
Published online Jan 16, 2026. doi: 10.12998/wjcc.v14.i2.116519
Figure 1
Figure 1 Visual field perimetry (octopus 101) demonstrating the preoperative and postoperative status. A: Left eye (Oculus Sinister) preoperatively, exhibiting significant visual field loss, particularly affecting the inferotemporal region; B: Left eye (Oculus Sinister) postoperatively, revealing marked improvement and substantial recovery of visual fields after surgical decompression.
Figure 2
Figure 2 T1-weighted magnetic resonance imaging scan showing a hyperintense fat-containing lesion within the left anterior clinoid process/sphenoid bone at the optic canal, causing extrinsic compression of the left optic nerve, consistent with an intraosseous lipoma.
Figure 3
Figure 3 Key surgical steps in extradural anterior clinoidectomy and optic nerve decompression. A: Extradural drilling of the anterior clinoid process using a diamond high-speed drill; B: Opening the optic canal with 1-mm Kerrison punch; C: Demonstration of extradural clinoidectomy and decompression of the optic canal; D: Opening the optic nerve dural sleeve; E: Use of a mini hook to verify complete optic nerve decompression intradural; F: Completely decompressed and free optic nerve, with adipose tissues (indicated by blue arrow) cleared away.
Figure 4
Figure 4 Postoperative axial T1-weighted magnetic resonance imaging demonstrating successful decompression of the left optic nerve canal and clearance of previously seen fat-containing lesion.