Published online Jun 25, 2025. doi: 10.5501/wjv.v14.i2.100986
Revised: December 4, 2024
Accepted: December 27, 2024
Published online: June 25, 2025
Processing time: 295 Days and 1.8 Hours
Rhabdomyolysis (RM) is characterized by disrupting muscle cells and releasing intracellular components into circulation. Some symptoms associated with RM include muscle weakness, discolored urine, and myalgia. RM can be caused by coronavirus disease 2019 (COVID-19) causing exaggerated immune response leading to muscle damage. Acute kidney injury (AKI), when presented with RM, leads to increased mortality. Examining RM-related AKI and its comparison to other AKI types in COVID-19 patients could improve the management of viral infections developing RM and AKI. RM potentially complicated COVID-19 infection course and is a major etiology of AKI. RM-related AKI had higher severity and mortality than other AKI types, with increased hypercoagulopathy and inflammatory markers. Findings also express procalcitonin use in follow-ups with severe COVID-19 patients. Study limitations include small sample size, absence of kidney biopsies, and focus on the first wave of the pandemic, which should be addressed in future research to generate accurate and relevant findings.
Core Tip: The study explored differences between rhabdomyolysis (RM)-related acute kidney injury (AKI) and other AKI types in coronavirus disease 2019 (COVID-19) patients. RM patients had high inflammation, procalcitonin, C-reactive protein, and ferritin. The prognosis of RM-related AKI was considered worse in comparison to developing AKI from other causes in COVID-19 patients. RM is a risk factor for AKI in COVID-19 patients. Hence, follow-ups are essential to monitor RM development. Study limitations include small sample size, absence of kidney biopsies, and focus on the 1st wave of the pandemic. Addressing these limitations leads to accurate result generation, potentially improving viral infection management for RM and AKI development.