From infection to invasion: The role of deleted in malignant brain tumors 1 in Helicobacter pylori-driven gastric cancer

doi:10.5306/wjco.v16.i10.108377

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Oct 24, 2025 (publication date) through Oct 27, 2025

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World Journal of Clinical Oncology

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2218-4333

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Clin Oncol. Oct 24, 2025; 16(10): 108377
Published online Oct 24, 2025. doi: 10.5306/wjco.v16.i10.108377

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Figure 1 Molecular mechanisms of Helicobacter pylori involved in gastric carcinogenesis. Copyright for the image which was created by author in Biorender (https://BioRender.com/1oa1hc) (Supplementary material). H. pylori: Helicobacter pylori; AKT: Protein kinase B; APC: Adenomatous polyposis coli; ASPP2: Apoptosis-stimulating protein of p53 2; CagA: Cytotoxic-associated gene A; CD8+ T cell: Cytotoxic T lymphocyte; ERK: Extracellular signal-regulated kinase; GSK3: Glycogen synthase kinase 3; PD-L1: Programmed death ligand 1; SALSA: Salivary scavenger and agglutinin; SHP2: Src homology-2 domain-containing phosphatase Shp2; T4SS: Type IV secretion system.

Citation: Negoi I. From infection to invasion: The role of deleted in malignant brain tumors 1 in Helicobacter pylori-driven gastric cancer. World J Clin Oncol 2025; 16(10): 108377
URL: https://www.wjgnet.com/2218-4333/full/v16/i10/108377.htm
DOI: https://dx.doi.org/10.5306/wjco.v16.i10.108377