Pathogenicity of Helicobacter pylori-associated gastric cancer

doi:10.5306/wjco.v16.i12.110909

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Dec 24, 2025 (publication date) through Dec 29, 2025

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World Journal of Clinical Oncology

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2218-4333

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Clin Oncol. Dec 24, 2025; 16(12): 110909
Published online Dec 24, 2025. doi: 10.5306/wjco.v16.i12.110909

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Figure 1 Multistep pathogenesis of Helicobacter pylori-induced gastric cancer. The diagram illustrates the progressive stages of Helicobacter pylori infection: From initial colonization and virulence factor expression (CagA and VacA) to activation of host immune responses (e.g., interleukin-8, tumor necrosis factor-alpha), chronic inflammation, tissue damage, atrophic gastritis, intestinal metaplasia, and eventual dysplasia leading to gastric cancer. IL: Interleukin; ROS: Reactive oxygen species; TNF-α: Tumor necrosis factor-alpha; H. pylori: Helicobacter pylori.

Citation: Ansari S, Ahmed N. Pathogenicity of Helicobacter pylori-associated gastric cancer. World J Clin Oncol 2025; 16(12): 110909
URL: https://www.wjgnet.com/2218-4333/full/v16/i12/110909.htm
DOI: https://dx.doi.org/10.5306/wjco.v16.i12.110909