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World J Clin Oncol. Oct 24, 2025; 16(10): 109385
Published online Oct 24, 2025. doi: 10.5306/wjco.v16.i10.109385
Figure 1
Figure 1 Circulating levels of inflammatory biomarkers in the pathogenesis of pancreatic cancer. Designed by the authors with the help of articles which are already cited in the references, (↑) increased levels (↓) decreased levels, (↨) not sure, (?) Not determined yet.
Figure 2
Figure 2 Haptoglobin causes pathogenesis in pancreatic cancer. Designed by the authors with the help of articles which are already cited in the references, haptoglobin phenotypes. Hp or Hpt: Haptoglobin.
Figure 3
Figure 3 Serum amyloid A causes pathogenesis in pancreatic cancer. Designed by the authors with the help of articles which are already cited in the references. SAA: Serum amyloid A; PDAC: Pancreatic ductal adenocarcinoma; CAFs: Cancer-associated fibroblasts; MAGUK: Membrane-associated guanylate kinase.
Figure 4
Figure 4 Fibrinogen causes pathogenesis in pancreatic cancer. Designed by the authors with the help of articles which are already cited in the references, pancreatic cancer. PC: Pancreatic cancer; Fbg/PNI: Fibrinogen to prognostic nutritional index.