Published online Dec 14, 2020. doi: 10.3748/wjg.v26.i46.7338
Peer-review started: September 9, 2020
First decision: September 29, 2020
Revised: October 9, 2020
Accepted: November 4, 2020
Article in press: November 4, 2020
Published online: December 14, 2020
Processing time: 95 Days and 23.6 Hours
Intrahepatic cholestasis in pregnancy (ICP) is the most common liver disease during pregnancy, and its cause and course of progression are still poorly understood.
Women with ICP have a significantly increased risk of preterm birth, stillbirth and admission to the neonatal unit for treatment. At present, there is no specific drug for the treatment of ICP in clinical practice.
We speculate that changes in serum metabolism caused by ICP may influence the intestinal tract circumstance and thus the intestinal microbiome. The gut microbiota may represent a mechanism by which ICP affects the health of pregnant women and fetuses. Our goal is to find the novel therapeutic strategies of ICP in the field of the gut microbiota.
The serum nontarget metabolomes from each group were determined. Amplification of the 16S rRNA V3-V4 region was performed using fecal samples from the disease and healthy groups. By comparing the differences in the microbiota and metabolite compositions between the two groups, the relationship between the gut microbiota and serum metabolites was also investigated.
ICP patients have critical differences in the intestinal microflora composition (beta diversity) from that in healthy controls. At the genus level, most of the bacteria depleted in ICP are able to produce short-chain fatty acids (e.g., Faecalibacterium, Blautia and Eubacterium hallii), while the bacteria enriched in ICP are associated with bile acid metabolism (e.g., Parabacteroides and Bilophila). Our results also showed that specific genera were associated with the serum metabolome.
The serum metabolome was significantly correlated with the gut microbiota, indicating that the gut microbiota plays an important role in the occurrence and development of ICP. Our findings suggest that the intestinal microbiome can be used as a therapeutic target to provide new strategies for the diagnosis and treatment of ICP.
Our group will study gut microbiota and metabolome changes before and after drug treatment to provide a new regimen for ICP treatment. In the future we will also conduct intervention studies using prebiotics, probiotics and synbiotics to promote the establishment of beneficial microbiota and investigate whether it can have a positive impact on the health of ICP patients.