Published online Jun 6, 2020. doi: 10.12998/wjcc.v8.i11.2255
Peer-review started: March 3, 2020
First decision: April 1, 2020
Revised: April 18, 2020
Accepted: April 28, 2020
Article in press: April 28, 2020
Published online: June 6, 2020
Processing time: 96 Days and 16.6 Hours
Hepatitis B virus (HBV) infection remains a global major public health problem. Chronic hepatitis B (CHB) patients generally have an impaired host immune response, which may be associated with persistently high viral load and subsequent T cell failure. Since peginterferon-alpha-2a (PEG-IFN) has direct antiviral and immunoregulatory effects, it has become one of the first choice drugs for the treatment of CHB. PEG-IFN has the properties of immune regulation, and the host immune status may affect the efficacy of PEG-IFN in the treatment of CHB.
Recently, many studies have shown that cytokines and chemokines may play a potential role in chronic viral hepatitis. We aimed to determine the correlation between cytokine/chemokine expression levels and response to PEG-IFN treatment in patients with CHB.
Our main purpose was to analyze the serum levels of cytokines in CHB patients receiving PEG-IFN treatment and responses to the therapy, and to assess the predictive value of the cytokines for the responses to PEG-IFN.
In total, 78 serum samples were prospectively collected with written informed consent from 26 CHB patients undergoing PEG-IFN therapy. A Luminex 200 analyzer and Cytokine Array I reagents were used to quantify 46 cytokines in the serum. The serum HBV DNA levels were assayed using a COBAS Amplicor/COBAS TaqMan HBV test, and HBeAb was measured by enzyme immunoassay.
A total of 26 patients (17 males and 9 females; mean age, 28.8.3 ± 4.161 years in virological responders group, and 27.82 ± 4.446 years in virological non-responders group) were enrolled in the study. The monokine induced by INF-γ (CXCL9) and serum interferon-inducible protein 10 (IP-10) levels at baseline were higher in the virological responders than in the non-virological responders and decreased during treatment. The CXCL9, IP-10, macrophage inflammatory protein 1d (MIP-1d), and thymus and activation-regulated chemokine (TARC) baseline levels exhibited the expected effects for interferon treatment. The area under the receiver operating characteristic curve values of CXCL9, IP-10, MIP-1d, and TARC for predicting the virological response were 0.787, 0.799, 0.787, and 0.77 (P = 0.01, 0.013, 0.01, and 0.021), respectively.
In conclusion, our results suggest that MIP-1d, CXCL9, CXCL6, IP-10, and TARC have predictive significance in interferon therapy. Cytokines are fundamental molecules in the complex signaling network of cell-mediated immunity. Analyses of the changes in cytokine expression patterns during treatment can help to understand the pathogenesis of CHB and predict treatment responses.
Further large and long-term follow-up studies are required to determine the predictive value of cytokines in CHB patients receiving PEG-IFN treatment. Besides, the differences in these factors between the response and non-response groups and their specific biological roles in HBV infection require further investigation.