Published online Aug 6, 2023. doi: 10.12998/wjcc.v11.i22.5204
Peer-review started: May 10, 2023
First decision: June 20, 2023
Revised: June 23, 2023
Accepted: July 7, 2023
Article in press: July 7, 2023
Published online: August 6, 2023
Processing time: 85 Days and 4.6 Hours
The treatment of hepatitis C with direct-acting antiviral agents (DAAs) produces a high rate of sustained virological response (SVR). But even after SVR, a certain number of patients develop cancer. Therefore, predicting the risk of carcinogenesis is important in such patients.
Both the age-male-albumin-bilirubin-platelets (aMAP) score and the velocity of shear waves (Vs) measured by shear wave elastography (SWE) have been shown to be useful for stratifying the risk of hepatocellular carcinoma (HCC) in hepatitis C who achieved SVR following DAAs therapy. We considered that combining the aMAP score with Vs improve the prediction of carcinogenic risk.
Objective of this study is to determine whether combining the aMAP score with Vs improves carcinogenic risk stratification in medium-to-high-risk hepatitis C patients.
Hepatitis C patients who achieved SVR with DAA therapy were enrolled. The medium-risk and high-risk groups with aMAP scores ≥ 50 at 12 wk (follow-up12) after treatment were divided into non-carcinogenic and carcinogenic groups. Clinical parameters in which significant differences were seen between non-carcinogenic and carcinogenic groups were taken as explanatory variables, and multiple regression analysis was performed with the presence or absence of carcinogenesis as the target variable. The diagnostic performances of clinical parameters for predicting the presence of HCC were evaluated using receiver-operating characteristic (ROC) curve analyses.
Multiple regression analysis was performed with carcinogenesis as the target variable and alanine aminotransferase, platelets, α-fetoprotein, Vs, and the Fib-4 index as explanatory variables; only Vs was found to be significant (P = 0.0296). The cut-off value for Vs calculated using the ROC curve for liver carcinogenesis was 1.53 m/s. When medium-risk and high-risk group people were stratified using this cut-off value, carcinogenesis was seen 2.0% from the group with Vs < 1.53 m/s 10.5% from the group with Vs ≥ 1.53 m/s.
In hepatitis C patients after SVR, a strategy of combining the aMAP score and Vs and stratifying the risk of carcinogenesis is more efficient than uniform surveillance of all patients.
Concentrated surveillance of patients selected for higher carcinogenesis risk will be more efficient than uniform surveillance of all patients after SVR.