Published online Jul 6, 2024. doi: 10.12998/wjcc.v12.i19.3824
Revised: May 7, 2024
Accepted: May 27, 2024
Published online: July 6, 2024
Processing time: 122 Days and 20.2 Hours
Affective disorders (AD) have been linked to inflammatory processes, although the underlying mechanisms of this relationship are still not fully elucidated. It is hypothesized that demographic, somatic, lifestyle, and personality variables pre
To identify biopsychosocial factors contributing to inflammation in AD measured with two parameters, C-reactive protein (CRP) and leukocytes.
This observational study investigated 186 hospital inpatients diagnosed with AD using demographic parameters, serum inflammatory markers, somatic variables, psychological questionnaires, and lifestyle parameters. Hierarchical regression an
Analyses showed that 33.8% of the variance of CRP was explained by body mass index and other somatic medication (e.g. anti-diabetics), age and education, and age of affective disorder diagnosis. For leukocytes, 20.1% of the variance was explained by smoking, diet, metabolic syndrome (MetS), and anti-inflammatory medication (e.g. non-steroidal anti-inflammatory drugs). Other psychiatric or behavioural variables did not reach significance.
Metabolic components seem important, with mounting evidence for a metabolic affective disorder subtype. Lifestyle modifications and psychoeducation should be employed to prevent or treat MetS in AD.
Core Tip: Somatic and psychological variables may affect inflammation in affective disorders (AD). In 186 inpatients with AD, C-reactive protein (CRP) and leukocytes were measured. CRP relates to somatic variables (e.g., body mass index), age, education, and age of diagnosis. Leukocytes relate to smoking, diet, and somatic variables (e.g., metabolic syndrome). Lifestyle modifications and psychoeducation on MetS is needed in individuals with AD to prevent symptom deterioration.