Published online Mar 6, 2023. doi: 10.12998/wjcc.v11.i7.1586
Peer-review started: October 27, 2022
First decision: January 9, 2023
Revised: January 14, 2023
Accepted: February 15, 2023
Article in press: February 15, 2023
Published online: March 6, 2023
Processing time: 126 Days and 0.2 Hours
It is not uncommon to develop autoimmune encephalitis and paraneoplastic neurological syndromes (PNS). 4 kinds of antibody-positive autoimmune paraneoplastic limbic encephalitis (PLE) have not been reported.
PNS are distant effects of cancer on the nervous system, rather than syndromes in which cancer directly invades and metastasizes to the nerves and/or muscle tissues. If the limbic lobe system of the brain is involved, this will result in PLE. The detection of patients with PNS is challenging since tumors that cause paraneoplastic neurologic disorders are often asymptomatic, obscure, and thus easily misdiagnosed or missed. Currently, single- or double-antibody-positive paraneoplastic marginal encephalitis has been reported. However, no cases of three or more-antibody-positive cases have been reported. Here, we report a case of PLE that is anti-collapsing response-mediator protein-5, anti-neuronal nuclear antibody-type 1, anti-aminobutyric acid B receptor, and anti-glutamate deglutase positive, and address relevant literature to improve our understanding of the disease.
This article reports on the management of a case of PLE with four positive antibodies, a review of the literature, in order to raise awareness among clinicians.
Core Tip: This patient mainly had persistent epilepsy as the main clinical manifestation, and a lumbar puncture examination showed anti-collapsing response mediator protein-5; anti-neuronal nuclear antibody-type 1; anti-aminobutyric acid B receptor; anti-glutamate deglutase positive, and further improvement of chest computed tomography and medical examination showed lung small cell carcinoma.