DNA methylation in gastric precancerous lesions: Molecular mechanisms and clinical translation

doi:10.5306/wjco.v17.i5.118073

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World Journal of Clinical Oncology

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2218-4333

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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Copyright: ©Author(s) 2026. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution-NonCommercial (CC BY-NC 4.0) license. No commercial re-use. See permissions. Published by Baishideng Publishing Group Inc.

World J Clin Oncol. May 24, 2026; 17(5): 118073
Published online May 24, 2026. doi: 10.5306/wjco.v17.i5.118073

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Figure 1 Molecular mechanisms of DNA methylation driving the progression of gastric precancerous lesions. H. pylori: Helicobacter pylori; EMT: Epithelial-mesenchymal transition.

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Figure 2 Clinical translation and application of DNA methylation biomarkers in gastric precancerous lesions. cfDNA: Cell-free DNA; AI: Artificial intelligence.

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Figure 3 Mechanisms of targeted intervention based on DNA methylation in gastric precancerous lesions. EBV: Epstein-Barr virus.

Citation: Zeng SH, Wu YQ, Zhang WJ, Wang YT, Xu L, Guo SJ. DNA methylation in gastric precancerous lesions: Molecular mechanisms and clinical translation. World J Clin Oncol 2026; 17(5): 118073
URL: https://www.wjgnet.com/2218-4333/full/v17/i5/118073.htm
DOI: https://dx.doi.org/10.5306/wjco.v17.i5.118073

Zeng SH, Wu YQ, Zhang WJ, Wang YT, Xu L, Guo SJ. DNA methylation in gastric precancerous lesions: Molecular mechanisms and clinical translation. World J Clin Oncol 2026; 17(5): 118073 [DOI: 10.5306/wjco.v17.i5.118073]

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