Update on the pathogenesis and clinical management of Helicobacter pylori gastric infection and associated diseases

doi:10.4291/wjgp.v16.i4.111432

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World Journal of Gastrointestinal Pathophysiology

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2150-5330

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Gastrointest Pathophysiol. Dec 22, 2025; 16(4): 111432
Published online Dec 22, 2025. doi: 10.4291/wjgp.v16.i4.111432

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Figure 1 Simplified representation of Helicobacter pylori adhesin-receptor interactions. Le^b: Lewis b antigen; sLe^a: Sialyl-Lewis an antigen; sLe^x: Sialyl-Lewis x antigen; CEACAMs: Carcinoembryonic Antigen-Related Cell Adhesion Molecules; T4SS: Type IV secretion system.

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Figure 2 Schematic summary of the underlying mechanisms to Helicobacter pylori-associated gastric diseases. IL-27: Interleukin 27; DupA: Duodenal ulcer promoting protein A; VacA: Vacuolating cytotoxin A; CagA: Cytotoxin-associated antigen A; MALT: Mucosa-associated lymphoid tissue.

Citation: Costa JMC, Aguiar CEO, Oliveira MMGL, Cenci Dietrich V, Lima PHM, Freire JPC, Lemos FFB, Queiroz DMM, de Melo FF. Update on the pathogenesis and clinical management of Helicobacter pylori gastric infection and associated diseases. World J Gastrointest Pathophysiol 2025; 16(4): 111432
URL: https://www.wjgnet.com/2150-5330/full/v16/i4/111432.htm
DOI: https://dx.doi.org/10.4291/wjgp.v16.i4.111432