BPG is committed to discovery and dissemination of knowledge
Case Report
Copyright ©The Author(s) 2025.
World J Cardiol. Dec 26, 2025; 17(12): 112389
Published online Dec 26, 2025. doi: 10.4330/wjc.v17.i12.112389
Figure 1
Figure 1 Laboratory testing. Complete set of labs including blood cell count, complete metabolic profile, cardiac testing, infectious testing, and rheumatologic testing.
Figure 2
Figure 2 Admission electrocardiogram. Normal sinus rhythm with frequent premature ventricular complexes. T wave repolarization changes in leads I and aVL.
Figure 3
Figure 3 Admission chest computed tomography. Note diffuse pulmonary nodules in all lung lobes and dilated main pulmonary trunk (blue arrow and orange arrow respectively).
Figure 4
Figure 4 Admission cardiac magnetic resonance imaging. Left ventricular (LV) dilation (arrows). Late gadolinium enhancement (LGE) in the basal to mid inferior segments of the LV and patchy mid-wall LGE in the basal inferolateral segment of the LV.
Figure 5
Figure 5 Positron emission tomography. Scans of heart and lungs. Variation in inflammation is demonstrable by fluorodeoxyglucose uptake differences. Uptake + in lungs (orange arrow) and – in heart (white arrow).
Figure 6
Figure 6 Follow up electrocardiogram. Electrocardiogram at 1.5 year after admission demonstrates resolution of arrhythmias since initial admission.
Figure 7
Figure 7 Follow up chest computed tomography. Chest computed tomography at 1 year after admission shows pulmonary nodules are improved since initial admission.