Published online Apr 15, 2024. doi: 10.4239/wjd.v15.i4.686
Peer-review started: December 20, 2023
First decision: January 10, 2024
Revised: January 19, 2024
Accepted: March 1, 2024
Article in press: March 1, 2024
Published online: April 15, 2024
Processing time: 113 Days and 20.6 Hours
The two-way relationship between periodontitis and type 2 diabetes mellitus (T2DM) is well established. Prolonged hyperglycemia contributes to increased periodontal destruction and severe periodontitis, accentuating diabetic complications. An inflammatory link exists between diabetic retinopathy (DR) and periodontitis.
Studies regarding this relation and the role of lipoprotein(a) [Lp(a)] and interleukin-6 (IL-6) in these conditions are scarce in the literature. This study assessed the proportion and severity of periodontitis and the correlation between periodontal inflamed surface area (PISA), and clinical attachment loss (CAL) with glycated hemoglobin (HbA1c), serum IL-6 and Lp(a).
(1) To determine and compare the proportion and severity of periodontitis in T2DM subjects with and without DR; (2) To assess the correlation between PISA and HbA1c, serum IL-6, and Lp(a) in T2DM subjects with and without DR; and (3) To assess the correlation between CAL and HbA1c, serum IL-6, and Lp(a) in T2DM subjects with and without DR.
The duration of the study was 18 months. In this study, 80 T2DM subjects (40 with DR and 40 without DR) were selected from the diabetic clinic of Department of Internal Medicine, Government Medical College, Calicut. They were divided into two groups based on the presence of DR as follows: Group I- T2DM with DR and Group II- T2DM without DR. Subjects were assessed with a detailed questionnaire regarding their socio-demographic characteristics, medical history, oral hygiene practice, history of diabetes and drug allergy. HbA1c, fasting plasma glucose and postprandial plasma glucose, serum IL-6, and Lp(a) were evaluated. Probing pocket depth, CAL, bleeding on probing, oral hygiene index-simplified, PISA and periodontal disease severity were determined. Diagnosis of DR was done by dilated fundoscopy.
The proportion of periodontitis in T2DM with DR and in T2DM without DR was 47.5% and 27.5% respectively. Severity of periodontitis, CAL, PISA, serum IL-6 and Lp(a) were higher in T2DM with DR group compared to T2DM without DR group. HbA1c was positively correlated with CAL (r = 0.351, P = 0.001), and PISA (r = 0.393, P ≤ 0.001) in study subjects. A positive correlation was found between PISA and IL6 (r = 0.651, P < 0.0001); PISA and Lp(a) (r = 0.59, P < 0.001); CAL and IL6 (r = 0.527, P < 0.0001) and CAL and Lp(a) (r = 0.631, P < 0.001) among study subjects.
The presence of DR may have contributed to the severity of periodontal destruction and periodontitis may have influenced the progression of DR.
Since a bidirectional link exists between periodontitis and diabetes mellitus, periodontal therapy should be included in the diabetes management. Proper periodontal care can help in improving glycemic control and prevent the progression of DR to some extent. A better understanding of the association between type 2 DR and periodontitis will help to create awareness among the public and to improve their overall quality of life.