Published online Aug 15, 2023. doi: 10.4251/wjgo.v15.i8.1451
Peer-review started: June 4, 2023
First decision: June 16, 2023
Revised: June 23, 2023
Accepted: July 7, 2023
Article in press: July 7, 2023
Published online: August 15, 2023
Processing time: 66 Days and 22.7 Hours
Type I gastric neuroendocrine neoplasms (gNENs) in chronic autoimmune gastritis (AIG) are increasingly being diagnosed, but no accurate data are available.
To date, no risk factors for malignant transformation of AIG into NENs have been identified. Their identification may significantly help to optimize endoscopic follow-up of AIG.
The main objective was to evaluate the incidence and prevalence of type I gNENs in a cohort of AIG patients. Secondly, the study aimed to identify potential risk factors for malignant transformation of AIG.
Patients with a histological diagnosis of AIG were enrolled between October 2020 and May 2022. Circulating levels of chromogranin A and gastrin were assessed at enrollment. Patients underwent regular endoscopic follow-up to detect gastric neoplastic lesions, enterochromaffin-like cell hyperplasia, and the development of gNEN.
Among 176 included patients, after 1032 person-years, 33 patients developed a total of 50 gNEN type I, with an incidence rate of 0.057 person-years, corresponding to an annual cumulative incidence of 5.7%. Gastrin levels were significantly higher in AIG patients who developed gNENs [median 992 pg/mL, [interquartile range (IQR) = 449–1500 vs 688 pg/mL, IQR = 423–1200[ (P = 0.03]). Calculated gastrin sensitivity and specificity were 90.9% and 1.4%, respectively, with an overall diagnostic accuracy of 30% and a calculated area under the gastrin receiver operating characteristic curve (AUROC or AUC) of 0.53.
Type I gNENs are a frequent complication of AIG. Hypergastrinemia plays a trophic role in the gastric mucosa, but due to its low specificity, it cannot be a noninvasive marker of early diagnosis of gNEN in AIG.
Further efforts are needed to identify effective strategies for individualizing endoscopic follow-up of AIG patients for early diagnosis and treatment of superimposed neuroendocrine lesions.