Published online Aug 15, 2022. doi: 10.4251/wjgo.v14.i8.1510
Peer-review started: March 21, 2022
First decision: April 25, 2022
Revised: May 8, 2022
Accepted: July 16, 2022
Article in press: July 16, 2022
Published online: August 15, 2022
Processing time: 142 Days and 12.7 Hours
Switching to a second line of systemic therapy will theoretically concern most patients with advanced hepatocellular carcinoma (HCC), especially after sorafenib. The strict selection criteria in phase III trials result in a lack of data for many patients from current practice. Inflammation acts as a powerful tumor promoter.
Two multi-targeted tyrosine kinase inhibitors (TKIs) [Regorafenib (REG), Cabozantinib (CBZ)] are currently the only available therapeutic options in France in this situation based on phase III trials after sorafenib. There are also no direct comparative studies between the "approved" second-line molecules or any predictive biomarker correlated with treatment activity.
To assess both efficacy and safety of REG and CBZ as second-line systemic treatment after sorafenib in a "real-life" study. To investigate the relevance of serum inflammation-related markers as predictive factors for tumor progression over time in this setting. The current lack of treatment-guiding biomarkers and the safety profile of TKIs are limiting factors for this sequencing.
This is an indirect propensity score-matched comparative study based on recent retrospective data recorded in three French centers. We focused on progression-free survival and disease control rates of patients treated with REG or CBZ, and on factors associated with tumor progression over time.
Both efficacy and safety of REG and CBZ are comparable in this real-life study, and CBZ is still a third-line therapeutic option. Elevated levels of pretherapeutic inflammation-related markers [C-reactive protein (CRP) serum level, neutrophil-to-lymphocyte ratio (NLR)] are associated with poorer survival by using TKIs as second-line treatment for HCC.
In light of the limited tumor control rate with TKIs and the positive results of first- (anti-programmed death ligand-1 + anti-vascular endothelial growth factor) and second-line (anti-human cytotoxic T-lymphocyte antigen-4 + anti-programmed death receptor-1) combination therapies, the therapeutic "landscape" of advanced HCC will be changed in the second-line setting. We propose a 2-mo online progression risk calculation based on CRP serum level, NLR, and aspartate aminotransferase level to estimate the disease course under ITKs treatment.
The tumor microenvironment plays a key role in the suppression of an effective lymphocyte response. TKIs exhibit anti-angiogenic and immunomodulatory properties. Combinations of TKIs and immune checkpoint inhibitors are currently being evaluated as second-line systemic therapy for HCC.