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©The Author(s) 2026.
World J Hepatol. Feb 27, 2026; 18(2): 114551
Published online Feb 27, 2026. doi: 10.4254/wjh.v18.i2.114551
Published online Feb 27, 2026. doi: 10.4254/wjh.v18.i2.114551
Figure 1 Translational framework for acrylamide exposure and prevention.
Acrylamide from common foods can trigger oxidative stress, DNA adducts, inflammation, and mitochondrial dysfunction. Monitoring tools (biosensors, omics, risk stratification) and preventive measures (standardized ginger extract, food-processing mitigation) act as complementary pillars; their integration enables stratified prevention and evidence-based public health. CYP2E1: Cytochrome P450 2E1; Nrf2: Nuclear factor erythroid 2-related factor 2; GSH: Glutathione; SOD: Superoxide dismutase; CAT: Catalase; NF-кB: Nuclear factor kappa-light-chain-enhancer of activated B cells.
- Citation: Massimi A, Massimi M. Ginger as a nutraceutical shield: Counteracting acrylamide-induced liver injury. World J Hepatol 2026; 18(2): 114551
- URL: https://www.wjgnet.com/1948-5182/full/v18/i2/114551.htm
- DOI: https://dx.doi.org/10.4254/wjh.v18.i2.114551
