Published online Oct 26, 2020. doi: 10.4252/wjsc.v12.i10.1196
Peer-review started: May 20, 2020
First decision: June 5, 2019
Revised: June 13, 2020
Accepted: August 16, 2020
Article in press: August 16, 2020
Published online: October 26, 2020
Processing time: 159 Days and 1.2 Hours
Inferior alveolar nerve (IAN) and lingual nerve (LN) injuries are complications of mandibular third molar extraction, which usually lead to numbness of the lips and taste disorder and eventually impose an extremely large burden on the patient’s life. Among all kinds of mesenchymal stem cells, dental pulp stem cells (DPSCs), derived from the neural crest, have been shown to have several similarities to neural cells. DPSCs may have a great potential for nerve repair.
A brand-new and effective stem cell based treatment for nerve repair and regeneration is urgently needed.
Nerve diseases and injuries, which are usually accompanied by motor or sensory dysfunction and disorder, impose a heavy burden upon patients and greatly reduce their quality of life. DPSCs, derived from the neural crest, have many characteristics that are similar to those of neural cells, indicating that they can be an ideal source for neural repair. In this study, we aimed to explore the potential roles and molecular mechanisms of DPSCs in crushed nerve recovery.
DPSCs were isolated, cultured, and identified by multilineage differentiation and flow cytometry. Western blot and immunofluorescent staining were applied to analyze the expression levels of neurotrophic proteins in DPSCs after neural induction. Then, we collected the secretions of DPSCs. We analyzed their effects on RSC96 cell proliferation and migration by CCK8 and transwell assays. Finally, we generated a sciatic nerve crush injury model in vivo and used the sciatic function index, walking track analysis, muscle weight, and hematoxylin & eosin (H&E) staining to further evaluate the nerve repair ability of DPSCs.
DPSCs highly expressed several specific neural markers, including GFAP, S100, Nestin, P75, and NF200, and were inclined toward neural differentiation. Furthermore, we also found that neural-induced DPSCs (N-DPSCs) could express neurotrophic factors, including NGF, BDNF, and GDNF. The secretions of N-DPSCs could enhance the proliferation and migration of Schwann cells. In vivo, both DPSC and N-DPSC implants alleviated gastrocnemius muscle atrophy. However, in terms of anatomy and motor function, as shown by H&E staining, immunofluorescent staining, and walking track analyses, the repair effects of N-DPSCs were more sustained, potent, and effective than those of DPSCs and the controls.
In summary, DPSCs might be an ideal cell source for nerve repair and regeneration, as they are inclined to undergo neural differentiation, express neurotrophic proteins after induction, and enhance SC proliferation after neural induction. Furthermore, N-DPSCs provide an effective and long-term treatment for crushed nerves with functional recovery and anatomical repair. Thus, DPSCs could be a promising therapeutic cell source for peripheral nerve repair and regeneration.
The application of DPSCs has several advantages, including ready accessibility, damage-free extraction from the donor’s body, few ethics issues, and low injection rates. We are able to isolate and culture dental pulp cells from extracted teeth and store them as seed cells for nerve repair, which can turn discarded clinical waste into a potential therapeutic resource. Our research has provided DPSCs as another possibility for nerve repair and regeneration.