Immune and metabolic cross-links in the pathogenesis of comorbid non-alcoholic fatty liver disease

doi:10.3748/wjg.v29.i4.597

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World Journal of Gastroenterology

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1007-9327

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Gastroenterol. Jan 28, 2023; 29(4): 597-615
Published online Jan 28, 2023. doi: 10.3748/wjg.v29.i4.597

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Figure 1 Risk factors and links of non-alcoholic fatty liver disease pathogenesis. NAFL: Non-alcoholic fatty liver; NASH: Non-alcoholic steatohepatitis.

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Figure 2 Cellular mechanisms of non-alcoholic fatty liver disease pathogenesis. DAMPs: Damage associated molecular patterns.

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Figure 3 Kupffer cell polarization. LPS: Lipopolysaccharide; FAO: Fatty acid oxidation; FAS: Fatty acid synthesis; IFN-γ: Interferon gamma; IL: Interleukin; iNOS: Inducible nitric oxide synthase; OXPHOS: Oxidative phosphorylation.

Citation: Kotlyarov S. Immune and metabolic cross-links in the pathogenesis of comorbid non-alcoholic fatty liver disease. World J Gastroenterol 2023; 29(4): 597-615
URL: https://www.wjgnet.com/1007-9327/full/v29/i4/597.htm
DOI: https://dx.doi.org/10.3748/wjg.v29.i4.597