Published online May 14, 2017. doi: 10.3748/wjg.v23.i18.3279
Peer-review started: December 30, 2016
First decision: January 19, 2017
Revised: February 3, 2017
Accepted: March 3, 2017
Article in press: March 3, 2017
Published online: May 14, 2017
Processing time: 137 Days and 14.3 Hours
To investigate wall shear stress (WSS) magnitude and distribution in cirrhotic patients with portal hypertension using computational fluid dynamics.
Idealized portal vein (PV) system models were reconstructed with different angles of the PV-splenic vein (SV) and superior mesenteric vein (SMV)-SV. Patient-specific models were created according to enhanced computed tomography images. WSS was simulated by using a finite-element analyzer, regarding the blood as a Newtonian fluid and the vessel as a rigid wall. Analysis was carried out to compare the WSS in the portal hypertension group with that in healthy controls.
For the idealized models, WSS in the portal hypertension group (0-10 dyn/cm2) was significantly lower than that in the healthy controls (10-20 dyn/cm2), and low WSS area (0-1 dyn/cm2) only occurred in the left wall of the PV in the portal hypertension group. Different angles of PV-SV and SMV-SV had different effects on the magnitude and distribution of WSS, and low WSS area often occurred in smaller PV-SV angle and larger SMV-SV angle. In the patient-specific models, WSS in the cirrhotic patients with portal hypertension (10.13 ± 1.34 dyn/cm2) was also significantly lower than that in the healthy controls (P < 0.05). Low WSS area often occurred in the junction area of SV and SMV into the PV, in the area of the division of PV into left and right PV, and in the outer wall of the curving SV in the control group. In the cirrhotic patients with portal hypertension, the low WSS area extended to wider levels and the magnitude of WSS reached lower levels, thereby being more prone to disturbed flow occurrence.
Cirrhotic patients with portal hypertension show dramatic hemodynamic changes with lower WSS and greater potential for disturbed flow, representing a possible causative factor of PV thrombosis.
Core tip: For portal hypertension, idealized portal vein (PV) modeling showed a significantly lower wall shear stress (WSS) in both sides of the PV and the occurrence of disturbed flow in the left wall of the PV. In addition, greater risk of disturbed flow was found for smaller PV-splenic vein (SV) angle and larger superior mesenteric vein-SV angle. In patient-specific models, WSS in cirrhotic patients with portal hypertension was markedly lower than that in healthy controls and disturbed flow was more likely to occur in the portal hypertension patients.