Role of nitric oxide in cerebral ischemia/reperfusion injury: A biomolecular overview

doi:10.12998/wjcc.v13.i10.101647

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Apr 6, 2025 (publication date) through Jan 10, 2025

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World Journal of Clinical Cases

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2307-8960

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Clin Cases. Apr 6, 2025; 13(10): 101647
Published online Apr 6, 2025. doi: 10.12998/wjcc.v13.i10.101647

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Figure 1 The biomolecular pathways of nitric oxide in brain damage after cerebral ischemia/reperfusion injury. eNOS: Endothelial nitric oxide synthase or type 3; nNOS: Neuronal nitric oxide synthase or type 1; iNOS: Inducible nitric oxide synthase or type 2); NO: Nitric oxide; NO₂: Nitrogen dioxide; ONOO^-: Peroxynitrite; PCD: Programmed cell death.

Citation: Anaya-Prado R, Canseco-Villegas AI, Anaya-Fernández R, Anaya-Fernandez MM, Guerrero-Palomera MA, Guerrero-Palomera C, Garcia-Ramirez IF, Gonzalez-Martinez D, Azcona-Ramírez CC, Garcia-Perez C, Lizarraga-Valencia AL, Hernandez-Zepeda A, Palomares-Covarrubias JF, Blackaller-Medina JH, Soto-Hintze J, Velarde-Castillo MC, Cruz-Melendrez DA. Role of nitric oxide in cerebral ischemia/reperfusion injury: A biomolecular overview. World J Clin Cases 2025; 13(10): 101647
URL: https://www.wjgnet.com/2307-8960/full/v13/i10/101647.htm
DOI: https://dx.doi.org/10.12998/wjcc.v13.i10.101647