Emerging treatments and current strategies for mineral, vascular, and bone disorders in chronic kidney disease

doi:10.5527/wjn.v15.i1.114146

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Mar 25, 2026 (publication date) through Mar 14, 2026

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World Journal of Nephrology

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2220-6124

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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Copyright: ©Author(s) 2026. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution-NonCommercial (CC BY-NC 4.0) license. No commercial re-use. See permissions. Published by Baishideng Publishing Group Inc.

World J Nephrol. Mar 25, 2026; 15(1): 114146
Published online Mar 25, 2026. doi: 10.5527/wjn.v15.i1.114146

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Figure 1 Schematic representation of phosphate handling and systemic effects in chronic kidney disease. Metal-based and resin-based phosphate binders act within the intestinal lumen to limit phosphate absorption. Persistent hyperphosphatemia contributes to vascular calcification, bone disorders, and cardiovascular complications (created in BioRender). NHE3: Sodium/hydrogen exchanger isoform 3; PTH: Parathyroid hormone.

Citation: Ilkun O, Jazayeri F, Kazory A. Emerging treatments and current strategies for mineral, vascular, and bone disorders in chronic kidney disease. World J Nephrol 2026; 15(1): 114146
URL: https://www.wjgnet.com/2220-6124/full/v15/i1/114146.htm
DOI: https://dx.doi.org/10.5527/wjn.v15.i1.114146