Published online Jul 19, 2023. doi: 10.5498/wjp.v13.i7.435
Peer-review started: April 12, 2023
First decision: May 12, 2023
Revised: May 18, 2023
Accepted: May 31, 2023
Article in press: May 31, 2023
Published online: July 19, 2023
Processing time: 97 Days and 9.8 Hours
Genetic factors are known to play a significant role in the development of panic disorder (PD). However, the impact of genetic variants on PD is still a subject of controversy.
γ-Aminobutyric acid (GABA) is an important neurotransmitter that inhibits brain activity. Previous reports have linked the glutamate decarboxylase 1 (GAD1) genetic variants to various psychiatric disorders, including bipolar disorder, schizophrenia, attention-deficit/hyperactivity disorder, and heroin dependence. However, few studies have examined the relationship between GAD1 and PD, particularly in Chinese populations.
The main objectives of this study were to examine the associations between GAD1 gene polymorphisms (rs1978340 and rs3749034) and PD risk, and to determine the effect of these polymorphisms on the severity of clinical symptoms, specifically respiratory symptoms, in individuals with PD.
The study included a total of 230 PD patients and 224 healthy controls. All participants underwent assessments for anxiety and panic symptom severity using the Hamilton Anxiety Rating Scale (HAM-A) and Panic Disorder Severity Scale (PDSS). The GAD1 gene polymorphisms (rs1978340 and rs3749034) were genotyped, and allele frequencies were analyzed.
The study findings revealed no significant differences in the genotype distributions or allele frequencies of GAD1 (rs1978340 and rs3749034) between the PD cases and the control group. Furthermore, the GAD1 gene polymorphisms (rs1978340 and rs3749034) did not exhibit a significant effect on the overall severity of PD. However, in relation to respiratory symptoms, PD patients with the GAD1 rs1978340 A/A genotype demonstrated significantly higher scores compared to those with the A/G or G/G genotype.
In conclusion, this study demonstrated that the A/A genotype of GAD1 rs1978340 is associated with increased severity of respiratory symptoms in individuals with PD. However, no significant associations were found between GAD1 gene polymorphisms and the risk of developing PD or the overall severity of the disorder.
Further research is needed to explore other potential genetic factors involved in the development and severity of PD. Additionally, investigating the underlying mechanisms through which GAD1 gene polymorphisms affect respiratory symptoms in PD patients could provide valuable insights for future studies.