Published online Aug 12, 2020. doi: 10.4291/wjgp.v11.i5.104
Peer-review started: January 17, 2020
First decision: April 9, 2020
Revised: April 24, 2020
Accepted: June 27, 2020
Article in press: June 27, 2020
Published online: August 12, 2020
Processing time: 199 Days and 9.7 Hours
Inflammatory bowel disease (IBD) causes systemic vascular inflammation. The increased risk of venous as well as arterial thromboembolic phenomena in IBD is well established. More recently, a relationship between IBD and atherosclerotic cardiovascular disease (ASCVD) has been postulated. Systemic inflammatory diseases, such as rheumatoid arthritis and systemic lupus erythematosus, have well characterized cardiac pathologies and treatments that focus on prevention of disease associated ASCVD. The impact of chronic inflammation on ASCVD in IBD remains poorly characterized. This manuscript aims to review and summarize the current literature pertaining to IBD and ASCVD with respect to its pathophysiology and impact of medications in order to encourage further research that can improve understanding and help develop clinical recommendations for prevention and management of ASCVD in patients with IBD.
Core tip: Chronic inflammation in patients with inflammatory bowel disease (IBD) leads to increased risk of atherosclerotic cardiovascular disease (ASCVD). However, the role and potential impact of IBD therapy in modifying ASCVD risk, and vice-versa, remains poorly understood. Herein, we highlight the importance of ASCVD as an extraintestinal manifestation of IBD, discuss the pathophysiology common to both diseases, and explore the role of non-traditional risk factors of ASCVD in IBD. We intend to identify avenues for further clinical and translational research that may help develop clinical recommendations for the management of ASCVD risk in patients with IBD.