Observational Study
Copyright ©The Author(s) 2020.
World J Diabetes. Nov 15, 2020; 11(11): 527-539
Published online Nov 15, 2020. doi: 10.4239/wjd.v11.i11.527
Figure 1
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Figure 1 Pathogenesis of type 1 diabetes-cellular crosstalk. CD: Celiac disease; TNF-α: Tumor necrosis factor-α; IL 1-β: Interleukin 1-β; NK: Natural killer cell.
Figure 2
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Figure 2 Associated autoimmune disorders in type 1 diabetes. The prevalence of associated glandular (black) and non-glandular (light grey) autoimmune diseases in patients with type 1 diabetes + autoimmune diseases, followed at the Johannes Gutenberg University Medical Center.
Figure 3
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Figure 3 Prevalence of autoantibodies in type 1 diabetes. The prevalence of autoantibodies in patients with type 1 diabetes (T1D) (light grey) and in patients with T1D + autoimmune diseases (black), followed at the Johannes Gutenberg University Medical Center. T1D: Type 1 diabetes; AID: Autoimmune diseases.
Figure 4
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Figure 4 Autoantibodies in relatives. Prevalence of autoantibodies in the relatives of patients with type 1 diabetes only (T1D) (light grey) and in the relatives of those with T1D + autoimmune diseases (AID, black). T1D: Type 1 diabetes; AID: Autoimmune diseases.