Published online Nov 15, 2020. doi: 10.4239/wjd.v11.i11.553
Peer-review started: July 18, 2020
First decision: August 9, 2020
Revised: August 22, 2020
Accepted: October 15, 2020
Article in press: October 15, 2020
Published online: November 15, 2020
Processing time: 117 Days and 20.8 Hours
Diabetic nephropathy (DN) is the main cause of chronic kidney disease and end-stage renal disease worldwide. Current treatment approaches for DN rely on angiotensin converting enzyme inhibitors or angiotensin II type 1 receptor blockers (ARBs) to control blood pressure (BP), reduce proteinuria, and delay chronic kidney disease progression. However, renin–angiotensin–aldosterone system (RAAS) blockade may increase the long-term risk for end-stage renal disease in patients with diabetes. Thus, identifying novel, effective treatments for DN beyond RAAS blockade is imperative.
Although available clinical trials have shown that endothelin receptor (ER) antagonists may be a novel and beneficial drug for DN, no consistent conclusions regarding their sufficient effectiveness and safety for patients with DN have been presented.
This meta-analysis aimed to assess the effectiveness and safety of ER antagonists among patients with DN.
We enrolled seven studies with six data sets and 5271 participants. The ER antagonists group showed a significantly greater reduction in albuminuria and more patients with 40% reduction in urinary albumin-to-creatinine ratio than the control group (P < 0.0001 and P = 0.02, respectively). Subgroup analysis for reductions in estimated glomerular filtration rate (eGFR) showed that for the middle-dosage subgroup, the ER antagonists group exhibited lower eGFR reduction than the control group (P < 0.00001; mean difference, 0.70 95%CI: 0.66, 0.74). Moreover, significant reductions in systolic blood pressure (SBP) and diastolic blood pressure (DBP) were observed in the invention group.
We enrolled seven studies with six data sets and 5271 participants. The ER antagonists group showed a significantly greater reduction in albuminuria and more patients with 40% reduction in urinary albumin-to-creatinine ratio than the control group (P < 0.0001 and P = 0.02, respectively). Subgroup analysis for reductions in eGFR showed that for the middle-dosage subgroup, the ER antagonists group exhibited lower eGFR reduction than the control group (P < 0.00001; mean difference, 0.70 95%CI: 0.66, 0.74). Moreover, significant reductions in SBP and DBP were observed in the invention group.
ER blockades combined with ACEI/ARBs may be an effective treatment to lower BP and reduce proteinuria in DN with declined eGFR. However, attention should be given to adverse events, including cardiac failure, anemia, and hypoglycemia, as well as serious adverse events.
ER blockade (optimal dosage) combined with ACEI/ARB may be an effective treatment for lowering BP, reducing proteinuria and delaying renal function progression in DN with declined eGFR. However, more long-term randomized controlled trials are still needed to validate the long-term effectiveness and safety of ER antagonists.