Published online May 15, 2019. doi: 10.4239/wjd.v10.i5.311
Peer-review started: March 4, 2019
First decision: April 13, 2019
Revised: April 17, 2019
Accepted: May 1, 2019
Article in press: May 1, 2019
Published online: May 15, 2019
Processing time: 76 Days and 20.9 Hours
The coexistence of sarcopenia and obesity has been termed as sarcopenic obesity (SO). Several studies have been conducted in order to determine any potential association between SO phenotype and type 2 diabetes (T2D). However, the available data are still contradictory and require further clarification.
To our knowledge no systematic review on the primary outcome related to the association between SO and T2D has been conducted yet to provide an unbiased interpretation of the evidence published to date.
We set out to systematically review the published literature with the aim of determining the prevalence of sarcopenia among adults with overweight and obesity and to investigate whether SO was associated with higher risk of T2D.
The review conformed to the Preferred Reporting Items for Systematic Review and Meta-Analyses guidelines, and data were collated by means of narrative synthesis and meta-analysis.
The prevalence of SO in adult with overweight and obesity is 43% in females and 42% in males, and the presence of this condition increases the risk of T2D by 38% with respect to those without SO.
A high prevalence of sarcopenia has been found among adults with overweight and obesity regardless of their gender, and this condition seems to be associated with a higher risk of T2D. The clinical implication of our findings is to raise awareness of the high prevalence of this phenotype in the overweight/obese population, and the importance of screening for SO in individuals affected by excess weight, since this condition seems to be strongly associated with T2D. However, our results need to be interpreted with caution with regard to the association between SO and the prevalence of T2D, since the cross-sectional design of the studies included in our systematic review indicates only associations between the two conditions and that does not provide information regard the causal relationships.
The current research indicates the need to design longitudinal studies to clarify the real effect of SO on the onset and progression of T2D. In other words, the available studies lack in evidence to determine if SO may lead to the onset or deterioration of T2D, since very few studies have longitudinally investigated the “real” effects of SO on health.