Gastric cancer: An epigenetic view

doi:10.4251/wjgo.v14.i1.90

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World Journal of Gastrointestinal Oncology

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1948-5204

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Gastrointest Oncol. Jan 15, 2022; 14(1): 90-109
Published online Jan 15, 2022. doi: 10.4251/wjgo.v14.i1.90

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Figure 1 Histone modifications in gastric cancer. Layers show different histone modifications. Blue panel: Modification types; Pink panel: Modified residues and catalytic enzymes; Green panel: Epigenetic alterations sites; Yellow panel: Biological functions regulated by histone modifications. HAT; Histone acetylase; HDAC: Histone deacetylase; HMT: Histone methyltransferase; HDM: Histone demethylase; UBE: Ubiquitin enzyme; DUB: Deubiquitinase.

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Figure 2 DNA methylation in gastric cancer. Aberrant methylation in promoter, shore area and gene body altered gene expression and involves in gastric carcinogenesis.

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Figure 3 Non coding RNA in gastric cancer. The major mechanism and biological function of lncRNA, miRNA and circRNA in gastric cancer.

Citation: Tang SY, Zhou PJ, Meng Y, Zeng FR, Deng GT. Gastric cancer: An epigenetic view. World J Gastrointest Oncol 2022; 14(1): 90-109
URL: https://www.wjgnet.com/1948-5204/full/v14/i1/90.htm
DOI: https://dx.doi.org/10.4251/wjgo.v14.i1.90