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Copyright: ©Author(s) 2026. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution-NonCommercial (CC BY-NC 4.0) license. No commercial re-use. See permissions. Published by Baishideng Publishing Group Inc.
World J Gastrointest Oncol. Jun 15, 2026; 18(6): 116858
Published online Jun 15, 2026. doi: 10.4251/wjgo.v18.i6.116858
Translational implications of the MAZ/UHRF1/ECRG4 axis in metastatic colorectal carcinoma
Eric Michael Kunz, Russell James Schoeller III, Joseph Patrick Carroll, Brandon Lucke-Wold
Eric Michael Kunz, Russell James Schoeller III, Joseph Patrick Carroll, Herbert Wertheim College of Medicine, Florida International University, Miami, FL 33199, United States
Brandon Lucke-Wold, Lillian S. Wells Department of Neurosurgery, University of Florida, Gainesville, FL 32608, United States
Author contributions: Kunz EM and Schoeller III RJ designed the overall concept and outline of the manuscript; Kunz EM and Lucke-Wold B contributed to the writing, editing the manuscript, and review of literature; Schoeller III RJ and Carroll JP contributed to the discussion and design of the manuscript; Kunz EM, Schoeller III RJ, Carroll JP, and Lucke-Wold B contributed to this paper, read and approved the final version of the manuscript to be published.
AI contribution statement: Grammarly and ChatGPT were used for language polishing and writing assistance for this manuscript.
Conflict-of-interest statement: All authors declare no conflict of interest in publishing the manuscript.
Corresponding author: Eric Michael Kunz, Doctorate Student, Herbert Wertheim College of Medicine, Florida International University, 11200 SW 8th Street, Miami, FL 33199, United States. ekunz005@med.fiu.edu
Received: November 24, 2025
Revised: December 21, 2025
Accepted: January 6, 2026
Published online: June 15, 2026
Processing time: 199 Days and 12.8 Hours
Core Tip

Core Tip: This manuscript identifies a novel metastatic signaling cascade in colorectal cancer in which Myc-associated zinc finger protein (MAZ) transcriptionally activates the epigenetic regulator ubiquitin-like with PHD and RING finger domains 1 (UHRF1), leading to hypermethylation and silencing of the tumor suppressor esophageal cancer-related gene 4 (ECRG4). This MAZ/UHRF1/ECRG4 axis drives epithelial – mesenchymal transition, invasion, and metastasis. Targeting MAZ or UHRF1 – or restoring ECRG4 expression – may offer a promising epigenetic therapeutic strategy for metastatic colorectal carcinoma.

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