Published online Apr 15, 2024. doi: 10.4251/wjgo.v16.i4.1213
Peer-review started: October 8, 2023
First decision: January 12, 2024
Revised: January 18, 2024
Accepted: February 23, 2024
Article in press: February 23, 2024
Published online: April 15, 2024
Processing time: 185 Days and 22.5 Hours
Portal vein thrombosis (PVT) is one of the complications of cirrhosis and one of the major public health concerns. PVT is found incidentally during the natural course of cirrhotic patients, and the formation of PVT is closely related to patient prognosis.
The identification of people at high risk for PVT is crucial for the prevention and treatment of PVT, therefore, it is necessary to develop an early prediction model of the probability of developing PVT in cirrhotic patients to guide clinical decision-making.
Development and validation of a nomogram and network calculator based on clinical blood inflammation markers for early identification of people at high risk of PVT.
By 1:1 propensity score matching, 572 eligible patients with cirrhosis were screened and their relevant clinical data were collected. Risk factors associated with the development of PVT were identified using the least absolute shrinkage and selection operator and multivariate logistic regression analysis. Variance inflation factor and correlation matrix plots tested for multicollinearity between variables. Finally, nomograms and network calculators predicting the risk of PVT occurrence were constructed and validated based on the independent risk factors for PVT.
A total of 572 patients with cirrhosis were included in this study. The final nine parameters identified as independent risk factors for PVT in cirrhosis were etiology, ascites, gastroesophageal varices (GOV), platelet count (PLT), D-dimer (D-D), portal vein diameter (PVD), portal vein velocity (PVV), aspartate aminotransferase-to-neutrophil ratio index (ANRI), and platelet-to-lymphocyte ratio (PLR). The area under the receiver operating characteristic of the constructed nomogram was 0.821 and 0.829 in the training and test groups, respectively. The calibration curves and DCA showed good clinical performance. Finally, the best threshold for the total score of the nomogram was 0.513. The sensitivity and specificity of the best threshold were 0.822 and 0.706, respectively.
Etiology, ascites, GOV, PLT, D-D, PVD, PVV, ANRI, and PLR were the independent risk factors for PVT in cirrhosis. The constructed nomogram had its excellent clinical performance.
In this article, we constructed a prediction model about the risk of developing PVT in the natural course of cirrhosis, and also confirmed that inflammatory markers have a good application value in the diagnosis of PVT. A convenient web-based calculator was also constructed to enable the assessment of the risk of developing PVT in patients with cirrhosis.