Published online Dec 15, 2023. doi: 10.4251/wjgo.v15.i12.2185
Peer-review started: August 15, 2023
First decision: October 9, 2023
Revised: October 16, 2023
Accepted: November 9, 2023
Article in press: November 9, 2023
Published online: December 15, 2023
Processing time: 117 Days and 4.6 Hours
Esophageal cancer is one of the most common cancers with high morbidity and mortality. It is unclear that paired-related homeobox 1 (PRRX1) induces epithelial-mesenchymal transition (EMT) in oesophageal cancer and the specific function of PRRX1 in oesophageal cancer metastasis.
This study is to assess the significance of PRRX1 expression and investigate the mechanism of EMT in oesophageal cancer metastasis.
One hundred primary oesophageal cancer tissue samples were collected from Thoracic Surgery and the human oesophageal squamous carcinoma cell lines EC109 and EC9706.
The expression of PRRX1 by the chemical test of immunohistochemical tissue is in the esophageal tumor tissue. The PRRX1 short hair holder RNA (SHRNA) or blank virus gene delivery system is transfected into the cells. Cell proliferation measurement, the formation of soft agar colonies, cell invasion and migration measurement, and animal research are used to observe the cell biological characteristics of internal and internal body. Mouse experimental testing and metastasis of esophageal tumors in the body. Immunfluorescence staining and protein marks analysis is used to detect protein expression of EMT labeling and Wnt/β-catenin. XAV939 and LiCl are used to change the activity of Wnt/β-catenin pathway.
PRRX1 is expressed in high levels in the specimen of esophageal cancer and is closely related to the metastasis and prognosis of the tumor metastasis and prognosis of patients with esophageal cancer. The regulation of PRRX1 may have a significant impact on cell proliferation, especially the migration and invasion of esophageal cancer cells. The expression of PRRX1 is closely related to EMT and Wnt/β-catenin. Further experiments, using shRNA to silence the expression of PRRX1 have the opposite effect. In addition, when PRRX1 was expressed, the inhibitory effect of XAV939 on WNT/β-catenin’s inhibitory has denied the effect of PRRX1 on EMT. When the expression of PRRX1 was downregulated, the Wnt/β-catenin pathway with LiCl impaired the effect on EMT.
In oesophageal cancer, PRRX1 is closely related to the metastasis and prognosis of tumor. PRRX1 can improve the ability to value and metastasize tumors. PRRX1 regulate EMT increases the ability of tumors. Further research found that PRRX1 regulates EMT through the Wnt/β-catenin signal.
The expression of PRRX1 is closely related to the metastasis and prognosis of esophageal cancer, which also shows that PRRX1 may become a potential esophageal cancer treatment target. Our studies have confirmed that PRRX1 is regulating EMT through WNT/β-catenin. More PRRX1 regulation mechanisms and downstream target areas need to be more in-depth research.