Published online Feb 27, 2024. doi: 10.4254/wjh.v16.i2.241
Peer-review started: October 22, 2023
First decision: December 8, 2023
Revised: December 21, 2023
Accepted: January 8, 2024
Article in press: January 8, 2024
Published online: February 27, 2024
Processing time: 127 Days and 18.2 Hours
Splenectomy has been considered an effective option to reverse thrombocytopenia in cirrhosis patients with splenomegaly. Thus, splenectomy have been widely used in Asia for the treatment of esophagogastric variceal hemonthsrrhage and hypersplenism caused by cirrhotic portal hypertension. However, splenectomy can increase the risk of portal vein thrombosis (PVT) at least 10 times. The incidence of PVT was 18.9%-57.0% after splenectomy, which was significantly higher than the natural incidence in patients with cirrhosis without surgery. PVT can induce or aggravate upper gastrointestinal bleeding, hepatic encephalopathy, and ascites, increase the risk of intestinal ischemia or intestinal necrosis, reduce the survival of patients and grafts after liver transplantation, and result in chronic cavernous transformation of the portal vein system in the long term.
Splenectomy plays an important role in the treatment of cirrhosis. Splenectomy is widely used for the treatment of esophagogastric variceal haemonthsrrhage and hypersplenism owing to cirrhotic portal hypertension. However, splenectomy can increase the risk of PVT at least 10 times. Our study aims to seek the risk factors of PVT after splenectomy and early sensitive indicators, to provide a predictive basis for early PVT and reduce the incidence of PVT.
To establish the risk factors for PVT after splenectomy and early sensitive indicators, to provide a predictive basis for early PVT.
A total of 45 patients with cirrhosis who underwent splenectomy were consecutively enrolled from January 2017 to December 2018. The incidence of PVT at 1 months, 3 months, and 12 months after splenectomy in patients with cirrhosis was observed. The hematological indicators, biochemical and coagulation parameters, and imaging features were recorded at baseline and at each observation point. The univariable, multivariable, receiver operating characteristic curve and time-dependent curve analyses were performed.
PVD ≥ 14.5 mm and monthsdel end-stage liver disease (MELD) > 10 were independent predictors of PVT at 1-months, 3-months, and 12-months after splenectomy. The patients with PVD ≥ 14.5 mm and/or MELD > 10 in preoperative, preoperative treatment of reducing portal vein pressure and improving liver function may help to reduce the incidence of PVT after splenectomy. However, monthsre large-scale studies will be needed to provide reliable and effective evidence for the specific time, drug selection and dosage of anticoagulants.
Portal vein diameter (PVD) ≥ 14.5 mm was independent predictors of PVT at 1-months, 3-months, and 12-months after splenectomy. End-stage liver disease score > 10 was independent predictors of PVT at 1-months, 3-months, and 12-months after splenectomy. The patients with PVD ≥ 14.5mm and/or end-stage liver disease score > 10 in preoperative, preoperative treatment of reducing portal vein pressure and improving liver function may help to reduce the incidence of PVT after splenectomy.
How to prophylactic anticoagulation therapy after splenectomy? Anticoagulant therapy of PVT should be explored.