Hydrogen sulfide attenuates gastric mucosal injury induced by restraint water-immersion stress via activation of KATP channel and NF-κB dependent pathway

Figure 1 Representative of the degree of gastric mucosal damage induced by restraint water-immersion stress. A: Representative of the degree of gastric mucosal damage in the control group; B: Representative of the degree of gastric mucosal damage in the physiological saline group; C: Representative of the degree of gastric mucosal damage in the NaHS group.

Figure 2 Gastric mucosal erosion index of the control, physiological saline and NaHS groups. ^eP < 0.001. GMEI: Gastric mucosal erosion index; PS: Physiological saline.

Figure 3 Representative of the degree of gastric mucosal damage in the NaHS group (A) and the glibenclamide + NaHS group (B).

Figure 4 Gastric mucosal erosion index in the NaHS group and the glibenclamide + NaHS group. Gl + NaHS group vs the NaHS group, ^eP < 0.001; Gl (100 μmol/kg body weight) plus NaHS group vs the Gl (150 μmol/kg body weight) + NaHS group, ^cP < 0.05. GMEI: Gastric mucosal erosion index; PS: Physiological saline; GI: Glibenclamide.

Figure 5 Representative of the degree of gastric mucosal damage in the NaHS group (A) and pyrrolidine dithiocarbamate + NaHS group (B).

Figure 6 Gastric mucosal erosion index in the NaHS group and PDTC + NaHS group. ^eP < 0.001. GMEI: Gastric mucosal erosion index; PDTC: Pyrrolidine dithiocarbamate; PS: Physiological saline.