Published online Oct 28, 2023. doi: 10.3748/wjg.v29.i40.5582
Peer-review started: August 7, 2023
First decision: August 28, 2023
Revised: August 31, 2023
Accepted: October 11, 2023
Article in press: October 11, 2023
Published online: October 28, 2023
Processing time: 81 Days and 6.4 Hours
Programmed death 1 (PD-1) and CD4+CD25+FoxP3+ expression in peripheral blood T-cells have been identified in multiple cancer types, but their variation during surgery and chemotherapy in gastric cancer remains elusive. Understanding this could illuminate tumor recurrence mechanisms and guide optimal anti-PD-1 antibody treatment strategies.
Despite known PD-1 and CD4+CD25+FoxP3+ expression in various cancers, the specific changes during surgery and chemotherapy, and their relationship in gastric cancer, remain undefined. This study seeks to shed light on these variations, potentially offering insights into tumor recurrence, immune evasion, and the clinical application of anti-PD-1 antibodies in gastric cancer.
The study aims to observe and analyze the expression characteristics of peripheral lymphocyte PD-1 and FoxP3+ regulatory T cells (FoxP3+Tregs) in gastric cancer patients, both prior to and following surgery or chemotherapy, to better understand their roles and implications in gastric cancer treatment.
In this study, 29 gastric cancer patients, post-D2 gastrectomy and undergoing chemotherapy, provided 10 mL peripheral blood samples during various perioperative phases. PD-1 expression was analyzed on specific lymphocyte subsets, with an additional 29 age-matched healthy donors serving as a control group.
The study found a significant elevation in PD-1 expression and FoxP3+ Tregs in gastric cancer patients, which decreased notably post-D2 gastrectomy. A positive correlation was identified between PD-1 expression and the number of activated FoxP3high Tregs in peripheral lymphocytes, especially during postoperative chemotherapy.
Alterations in PD-1 expression and regulatory T cell activation during gastric cancer treatment could provide valuable insights for understanding tumor immune evasion. These findings may also influence the clinical application of anti-PD-1 antibodies in gastric cancer therapy.
The changes observed in PD-1 expression and regulatory T cell activation during gastric cancer treatments pave the way for deeper exploration into tumor immune evasion mechanisms. These findings could also shape the future clinical application and optimization of anti-PD-1 antibodies in treating gastric cancer.