Published online Nov 28, 2017. doi: 10.3748/wjg.v23.i44.7830
Peer-review started: September 23, 2017
First decision: October 11, 2017
Revised: October 14, 2017
Accepted: October 27, 2017
Article in press: October 27, 2017
Published online: November 28, 2017
Processing time: 65 Days and 12.8 Hours
Many studies strongly suggest that signals, including bacterial DNA, from colonizing microbes greatly alter host local immune system in the gut. Bacterial cells do not contact with enterocytes in normal physiological status. They might release DNA into the mucus layer to influence host innate immune cell through specific receptors, like Toll-like receptor 9. Evidence supporting this hypothesis is needed.
This research investigated the existence of extracellular bacterial DNA (eDNA) in the mouse gut mucus layer, their resource, and immune modulatory function. There were differences in DNA’s immuno-stimulatory properties among different bacteria as reported by other researchers. Therefore, host immune response would be modulated by targeted change of DNA releasing bacteria in the mucus through specific medicine or food components.
This study aimed to confirm the existence of bacterial eDNA in the mouse gut mucus layer, and to identify bacterial genera that release them. Immuno-stimulatory properties of eDNA were also studied in vitro. This provided basic knowledge about bacteria and host interaction through bacterial DNA and related signal pathways. This will also promote nutritional strategy development to modulate local immune response through changing DNA releasing microbiota.
Bacterial eDNA in the mucus layer and crypts was visualized by TOTO-1 staining. Small intestinal mucosal microbiota and eDNA were analyzed using T-RFLP and Illumina MiSeq amplicon sequencing. Immuno-stimulatory effects of microbiota and eDNA were determined after incubation with mouse RAW264.7 macrophages.
TOTO-1 iodide staining confirmed existence of eDNA in the mucus layer. The composition of the eDNA was significantly different from that of the intracellular DNA (iDNA). The eDNA sequences came mainly from Gram-negative bacteria of Bacteroidales S24-7. The eDNA induced significantly lower TNF-α/IL-10 and IL-6/IL-10 ratios in LPS stimulated RAW264.7 cells than iDNA. This is the first report related to bacteria genus responsible for DNA release in the gut mucus layer.
Our results indicated that eDNA was located in the intestinal mucus layer. The eDNA was degraded bacterial genomic DNA mainly released by Gram-negative bacteria especially Bacteroidales S24-7. They showed decreased pro-inflammatory activity compared with total gut flora genomic DNA.
Further studies are needed to clarify the specific bacterial species/strains that release eDNA, and its relationship with the gut immune response, especially the production of antimicrobial peptides in Paneth cells of the small intestinal crypt.