Liver Cancer
Copyright ©The Author(s) 2003. Published by Baishideng Publishing Group Inc. All rights reserved.
World J Gastroenterol. Sep 15, 2003; 9(9): 1946-1949
Published online Sep 15, 2003. doi: 10.3748/wjg.v9.i9.1946
Pathomorphological study on location and distribution of Kupffer cells in hepatocellular carcinoma
Kai Liu, Xu He, Xue-Zhong Lei, Lian-San Zhao, Hong Tang, Li Liu, Bing-Jun Lei
Kai Liu, Xu He, Xue-Zhong Lei, Lian-San Zhao, Hong Tang, Li Liu, Bing-Jun Lei, Division of Molecular Biology of Infectious Disease Key Laboratory of Biotherapy of Human Disease, Ministry of Education, West China Hospital of Sichuan University, Chengdu 610041, Sichuan Province, China
Author contributions: All authors contributed equally to the work.
Supported by the National Natural Science Foundation of China, No. 39670812
Correspondence to: Lian-San Zhao, Division of Molecular Biology of Infectious Disease Key Laboratory of Biotherapy of Human Disease, Ministry of Education, West China Hospital of Sichuan University, Chengdu 610041, Sichuan Province, China
Telephone: +86-28-85422635 Fax: +86-28-85422113
Received: March 20, 2003
Revised: April 5, 2003
Accepted: April 14, 2003
Published online: September 15, 2003
Abstract

AIM: To clarify the location and distribution of Kupffer cells in hepatocellular carcinoma (HCC), and to investigate their role in hepatocarcinogenesis.

METHODS: Kupffer cells were immunohistochemically stained by streptavadin-peroxidase conjugated method (S-P). The numbers of Kupffer cells in cancerous, para-cancerous and adjacent normal liver tissues of 48 HCCs were comparatively examined.

RESULTS: The mean number of Kupffer cells in cancerous, para-cancerous and adjacent normal liver tissues was 12.7 ± 6.8, 18.1 ± 8.2 and 18.9 ± 7.9 respectively. The number of Kuppfer cells in cancerous tissues was significantly lower than that in para-cancerous tissues (t = 2.423, P < 0.05) and adjacent normal liver tissues (t = 2.521, P < 0.05). As tumor size increased, the number of Kupffer cells in cancerous tissues significantly decreased (F = 4.61, P < 0.05). Moreover, there was also a significant difference in the number of Kupffer cells among well-differentiated, moderately-differentiated and poorly-differentiated cases(F = 4.49, P < 0.05).

CONCLUSION: This study suggests that decrease of Kupffer cells in HCCs may play an important role in the carcinogenesis of HCC, the number of Kupffer cells in HCC is closely related to the size and differentiation grade of the tumor.

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