Liver Cancer
Copyright ©The Author(s) 2003. Published by Baishideng Publishing Group Inc. All rights reserved.
World J Gastroenterol. Feb 15, 2003; 9(2): 262-266
Published online Feb 15, 2003. doi: 10.3748/wjg.v9.i2.262
Potent inhibition of angiogenesis and liver tumor growth by administration of an aerosol containing a transferrin-liposome-endostatin complex
Xi Li, Geng-Feng Fu, Yan-Rong Fan, Chan-Fu Shi, Xin-Juan Liu, Gen-Xing Xu, Jian-Jun Wang
Xi Li, Geng-Feng Fu, Gen-Xing Xu, Jian-Jun Wang, School of Life Science, Nanjing University, 22 Hankou Road, Nanjing 210093, Jiangsu Province, China
Yan-Rong Fan, Chan-Fu Shi, Xin-Juan Liu, Gen-Xing Xu, Nanjing Military Medical College of the Second Military Medical University, 2 Maqun Road, Nanjing 210049, Jiangsu Province, China
Author contributions: All authors contributed equally to the work.
Supported by Jiangsu Natural Science Fund (China), BK20000001, the National Natural Science Foundation of China, 30070250 and the “985 Project” from Nanjing University
Correspondence to: Gen-Xing Xu, P.O.Box 4901, 2 Maqun Road, Nanjing 210049, Jiangsu Province, China. genxingx@yahoo.com.cn
Telephone: +86-25-4364753 Fax: +86-25-4350162
Received: July 31, 2002
Revised: August 4, 2002
Accepted: August 16, 2002
Published online: February 15, 2003
Abstract

AIM: To obtain an efficient delivery system for transporting endostatin gene to mouse liver tumor xenografts by administration of aerosol.

METHODS: Recombinant plasmid pcDNA3.0/endostatin containing human endostatin gene together with signal peptide from alkaline phosphatase were transferred into human umbilical vein endothelial cell (HUVEC) by transferrin (TF)-liposome-endostatin complex. Western blot was used to detect the expression of human endostatin in transfected HUVEC cells and its medium. After the tumor-bearing mice were administrated with TF-liposome-endostatin complex, the lung tissue was analyzed by immunohistochemical method for expression of endostatin and the tumors were treated with CD-31 antibody to detect the density of microvesseles in tumor tissues. The inhibition of tumor growth was estimated by the weight of tumors from groups treated with different doses of TF-liposome-endostatin complex. DNA fragmentation assay was used to detect the apoptosis of the cells from primary liver tumor.

RESULTS: Western blot analysis and immunohistochemical method confirmed the expression of endostatin protein in vitro and in vivo. After the tumor sections were treated with CD-31 antibody, the positive reaction cells appeared brown while the negative cells were colorless. The positively stained area of the TF-liposome-endostatin treated group was significantly smaller (P < 0.01, 645.8 ± 5.2 μm2) than that of the control group (1325.4 ± 98.5 μm2). The data showed a significant inhibition of angiogenesis. After administration of TF-liposome-endostatin, comparing with the control group administrated with TF-liposome-pcDNA3.0, liver tumor growth in the mice treated with 50, 250 and 500 mg DNA/kg was inhibited by 36.6%, 40.8%, and 72.8%, respectively (P < 0.01). And a typical DNA fragmentation of apoptosis was found in the cells from tumor tissues of the mice treated with TF-liposome-endostatin but none in the control group.

CONCLUSION: Endostatin gene could be efficiently transported into the mice with TF-liposome-DNA delivery system by administration of aerosol. TF-liposome-mediated endostatin gene therapy strongly inhibited angiogenesis and the growth of mouse xenograft liver tumors. It also could promote the development of apoptosis of tumors without direct influence on tumor cells.

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