Viral Hepatitis
Copyright ©The Author(s) 2003. Published by Baishideng Publishing Group Inc. All rights reserved.
World J Gastroenterol. Nov 15, 2003; 9(11): 2474-2479
Published online Nov 15, 2003. doi: 10.3748/wjg.v9.i11.2474
Establishment of a cell-based assay system for hepatitis C virus serine protease and its primary applications
Hong-Xia Mao, Shui-Yun Lan, Yun-Wen Hu, Li Xiang, Zheng-Hong Yuan
Hong-Xia Mao, Shui-Yun Lan, Yun-Wen Hu, Li Xiang, Zheng-Hong Yuan, Key Laboratory of Medical Molecular Virology, Shanghai Medical College, Fudan University, Shanghai 200032, China
Author contributions: All authors contributed equally to the work.
Supported by the National Natural Science Foundation of China, No. 39870010, No. 39825501, the Shanghai Scientific Development Foundation, No. 00QMH1404 and the State 973 Program of China, No. G1999054105
Correspondence to: Zheng-Hong Yuan, Key Laboratory of Medical Molecular Virology, Shanghai Medical College, Fudan University, 138 Yi Xue Yuan Road, Shanghai 200032, China. zhyuan@shmu.edu.cn
Telephone: +86-21-64161928 Fax: +86-21-64227201
Received: March 10, 2003
Revised: June 1, 2003
Accepted: June 12, 2003
Published online: November 15, 2003
Abstract

AIM: To establish an efficient, sensitive, cell-based assay system for NS3 serine protease in an effort to study further the property of hepatitis C virus (HCV) and develop new antiviral agents.

METHODS: We constructed pCI-neo-NS3/4A-SEAP chimeric plasmid, in which the secreted alkaline phosphatase (SEAP) was fused in-frame to the downstream of NS4A/4B cleavage site. The protease activity of NS3 was reflected by the activity of SEAP in the culture media of transient or stable expression cells. Stably expressing cell lines were obtained by G418 selection. Pefabloc SC, a potent irreversible serine protease inhibitor, was used to treat the stably expressing cell lines to assess the system for screening NS3 inhibitors. To compare the activity of serine proteases from 1b and 1a, two chimeric clones were constructed and introduced into both transient and stable expression systems.

RESULTS: The SEAP activity in the culture media could be detected in both transient and stable expression systems, and was apparently decreased after Pefabloc SC treatment. In both transient and stable systems, NS3/4A-SEAP chimeric gene from HCV genotype 1b produced higher SEAP activity in the culture media than that from 1a.

CONCLUSION: The cell-based system is efficient and sensitive enough for detection and comparison of NS3 protease activity, and screening of anti-NS3 inhibitors. The functional difference between NS3/4A from 1a and 1b subtypes revealed by this system provides a clue for further investigations.

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