Liver Cancer
Copyright ©The Author(s) 2003. Published by Baishideng Publishing Group Inc. All rights reserved.
World J Gastroenterol. Nov 15, 2003; 9(11): 2428-2432
Published online Nov 15, 2003. doi: 10.3748/wjg.v9.i11.2428
Segregation analysis of hepatocellular carcinoma in a moderately high-incidence area of East China
Ru-Lin Cai, Wei Meng, Hong-Yan Lu, Wen-Yao Lin, Feng Jiang, Fu-Min Shen
Ru-Lin Cai, Wei Meng, Feng Jiang, Fu-Min Shen, Department of Epidemiology, School of Public Health, Fudan University, Shanghai 200032, China
Hong-Yan Lu, Wen-Yao Lin, Haimen City Anti-epidemic Station, Haimen 226100, Jiangsu Province, China
Author contributions: All authors contributed equally to the work.
Supported by the National Natural Science Foundation of China, No. 39930160
Correspondence to: Dr. Wei Meng, Department of Epidemiology, School of Public Health, Fudan University, Shanghai 200032, China. wmeng@shmu.edu.cn
Telephone: +86-21-54237767 or 7710
Received: December 28, 2002
Revised: February 2, 2003
Accepted: February 19, 2003
Published online: November 15, 2003
Abstract

AIM: To explore the mode of inheritance of hepatocellular carcinoma (HCC) in a moderately high-incidence area of East China.

METHODS: A pedigree survey was conducted in 210 families (3315 individuals) ascertained through 210 HCC probands in Haimen, Jiangsu Province. Simple segregation analysis was conducted using SEGRANB software. The probability of ascertainment (π), segregation ratio (p), and the proportion of sporadic cases (x) were estimated. Complex segregation analysis was performed using the REGTL program of S.A.G.E. Models were fitted on the data of 3212 individuals that allowed for personal HBsAg status and variable age of onset in REGTL program.

RESULTS: The estimate of segregation ratio was 0.191 by SEGRANB. The probability of ascertainment was 0.0266, and the proportion of sporadic cases was 0.465. The results of complex segregation analysis showed that Mendelian autosomal recessive inheritance of a major gene that influenced the age of onset distribution of HCC, provided the best fit to the data. In the best-fitting recessive model, the frequency of the disease allele was 0.11138. HBsAg seropositive status would significantly increase the risk of developing HCC.

CONCLUSION: These results suggest that at least one major gene is involved in the genetic predisposition to develop HCC at an earlier age of onset. The seropositive HBsAg status can significantly increase the risk of developing HCC, which provides strong support for the interaction between genetic and environmental risk factors.

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