Basic Research
Copyright ©The Author(s) 2002. Published by Baishideng Publishing Group Inc. All rights reserved.
World J Gastroenterol. Apr 15, 2002; 8(2): 346-349
Published online Apr 15, 2002. doi: 10.3748/wjg.v8.i2.346
Nuclear factor κB activity in patients with acute severe cholangitis
Jian-Ping Gong, Chong-An Liu, Chuan-Xin Wu, Sheng-Wei Li, Yu-Jun Shi, Xu-Hong Li
Jian-Ping Gong, Chong-An Liu, Chuan-Xin Wu, Sheng-Wei Li, Yu-Jun Shi, Xu-Hong Li, Department of General Surgery, The Second College of Clinical Medicine & the Second Affiliated Hospital of Chongqing University of Medical Science, 74 Linjiang Road, Central District, Chongqing 400010, China
Author contributions: All authors contributed equally to the work.
Supported by the National Natural Science Foundation of China, No.39970719, 30170919
Correspondence to: Dr. Jian-Ping Gong, Department of General Surgery, The Second College of Clinical Medicine & the Second Affiliated Hospital of Chongqing University of Medical Science, 74 Linjiang Road, Central District, Chongqing 400010, China. gongjianping11@hotmail.com
Telephone: +86-23-63766701 Fax: +86-23-63822815
Received: September 14, 2001
Revised: October 15, 2001
Accepted: October 22, 2001
Published online: April 15, 2002
Abstract

AIM: To determine the NF-κB activity in peripheral blood mononuclear cells (PBMC) in patients with acute cholangitis of severe type (ACST) and correlate the degree of NF-κB activation with severity of biliary tract infection and clinical outcome.

METHODS: Twenty patients with ACST were divided into survivor group (13 cases) and nonsurvivor group (7 cases). Other ten patients undergoing elective gastrectomy or inguinal hernia repair were selected as control group. Peripheral blood samples were taken 24 h postoperatively. PBMC were separated by density gradient centrifugation, then nuclear proteins were isolated from PBMC, and Electrophoretic Mobility Shift Assay (EMSA) used determined. The results were quantified by scanning densitometer of a Bio-Image Analysis System and expressed as relative optical density (ROD). The levels of TNF-α, IL-6, and IL-10 in the plasma of patients with ACST and healthy control subjects were determined by using an enzyme-linked immunoassay (ELISA).

RESULTS: The NF-κB activity was 5.02 ± 1.03 in nonsurvivor group, 2.98 ± 0.51 in survivor group and 1.06 ± 0.34 in control group. There were statistical differences in three groups (P < 0.05). The levels of TNF-α and IL-6 in plasma were (498 ± 53) ng·L-1 and (587 ± 64) ng·L-1 in nonsurvivor group, (284 ± 32) ng·L-1 and (318 ± 49) ng·L-1 in survivor group and (89 ± 11) ng·L-1 and (102 ± 13) ng·L-1 in control group. All patients with ACST had increased levels of TNF-α and IL-6, which were manyfold greater than those of control group, and there was an evidence of significantly higher levels in those of nonsurvivor group than that in survivor group (P < 0.05). The levels of IL-10 in plasma were (378 ± 32) ng·L-1, (384 ± 37) ng·L-1 and (68 ± 11) ng·L-1 in three groups, respectively. All patients had also increased levels of IL-10 when compared with control group (P < 0.05), but the IL-10 levels were not significantly higher in nonsurvivors than in survivors (P > 0.05).

CONCLUSION: NF-κB activity in PBMC in patients with ACST increases markedly and the degree of NF-κB activation is correlated with severity of biliary tract infection and clinical outcome.

Keywords: $[Keywords]