Original Research
Copyright ©The Author(s) 2001. Published by Baishideng Publishing Group Inc. All rights reserved.
World J Gastroenterol. Aug 15, 2001; 7(4): 547-550
Published online Aug 15, 2001. doi: 10.3748/wjg.v7.i4.547
A study on pathogenicity of hepatitis G virus
Jia-Zhang Xu, Zhi-Guo Yang, Mei-Zhao Le, Mao-Rong Wang, Chang-Lun He, Yun-Hua Sui
Jia-Zhang Xu, Zhi-Guo Yang, Mao-Rong Wang, Chang-Lun He, Yun-Hua Sui, Institute of Hepatology, the 81st Hospital of PLA, Nanjing 210002, Jiangsu Province, China
Mei-Zhao Le, Department of Pathology, the 81st Hospital of PLA, Nanjing 210002, Jiangsu Province, China
Author contributions: All authors contributed equally to the work.
Supported by the Science Foundation of Jiangsu Province, No. BK97173
Correspondence to: Jia-Zhang Xu, Institute of Hepatology, the 81st Hospital of PLA, Nanjing 210002, Jiangsu Province, China
Received: March 13, 2001
Revised: April 27, 2001
Accepted: May 4, 2001
Published online: August 15, 2001
Abstract

AIM: To study the pathogenicity of hepatitis G virus (HGV) and observe the genesis and pathological process of hepatitis G.

METHODS: HGV-RNA in serum was detected by RT-PCR assay. The immunohistochemical assays of liver tissue were performed with HGV monocoloned antibody (McAb) expressed from the region of HGV NS5 nucleic acid sequence. The clinical and pathological data of 52 patients with hepatitis G were discussed. In animal experiment, the Chinese Rhesus monkeys were infected with the serum of a patient with HGV infection. And the dynamic changes in serology and liver histology of animals were observed.

RESULTS: One hundred and fifty-four patients with HGV-RNA positive were selected from 1552 patients with various kinds of hepatitis. Of 154 patients with HGV infection, 52 were infected with HGV only, which accounted for 33.8% (52/154) and 102 with positive HGV-RNA were super-infected with other hepatitis viruses, which accounted for 66.2% (102/154). The clinical and pathological observation showed that the acute and chronic hepatitis could be induced by HGV. The slight abnormality of transaminases ALT and AST in serum of monkeys lasted nearly 12 months and histological results showed a series of pathological changes.

CONCLUSION: HGV is a hepatotropic virus and has pathogenicty.

Keywords: hepatitis virus G; pathogenicity