Published online Jun 15, 1999. doi: 10.3748/wjg.v5.i3.228
Revised: February 20, 1999
Accepted: February 25, 1999
Published online: June 15, 1999
AIM: To investigate the role of apolipoprotein E (apoE) polymorphism in the lithogenesis of gallstone and the hereditary pathogenesis of the disease.
METHODS: Polymerase chain reaction (PCR) was used to study apoE phenotypes and allele frequencies in patients with gallstones and control, and the fasting serum lipids of subjects were also measured by enzymatic methods.
RESULTS: The levels of triglyceride (TG) and very low density lipoprotein cholesterol (VLDL-C) were much higher in E2/3 patients than that in E2/3 control. E3/3 patients were accompanied with remarkably low levels of high density lipoprotein cholesterol(HDL-C) and its subforms. But in E3/4 patients there were only slight changes in levels of VLDL-C and low density lipoprotein cholesterol (LDL-C).
CONCLUSION: Different apoE phenotype patients with gallstones have different cheracteristics of dyslipidemia and the average level of serum lipids in patients with gallstones are higher than subjects without gallstones in the same apoE gene phenotype. ε2 allele is possibly one of the dangerous factors in the lithogenesis of chole-cystolithiasis.