Published online Feb 21, 2025. doi: 10.3748/wjg.v31.i7.97599
Revised: November 25, 2024
Accepted: December 18, 2024
Published online: February 21, 2025
Processing time: 230 Days and 3.9 Hours
Radiation enteritis is a common complication of radiation therapy in which the surrounding normal intestinal tissue is damaged by ionising radiation, and there is no standard pharmacological prophylaxis or treatment regimen available. Mesenchymal stem cell transplantation can be used for radiation protection and the treatment of acute radiation injury, but its therapeutic mechanism of action remains unclear.
To investigate the protective effects of autologous bone marrow-derived me
A model of acute radioactive enteritis was established in dogs by applying abdominal intensity-modulated radiation at a single X-ray dose of 12 Gy. ABMSCs were transplanted into the mesenteric artery with the technology of femoral artery puncture and DSA imaging two days after radiation. Visual and histopathological changes of the experimental dogs were observed. Different kinds of cytokines from intestinal samples were tested using Quantibody Canine Cytokine Array method. Enzyme-linked immunosorbent assay (ELISA) was also used to evaluate the cytokines changes in serum.
The ABMSCs group showed significant improvements in survival status compared with the blank and saline treatment groups. Histological observations revealed that the former had lower histological scores than the later after treatment (P < 0.05). Compared to the control groups, interleukin (IL)-10 and monocyte chemotactic protein (MCP)-1 from intestinal samples showed a remarkable increase and ELISA of serum samples proved higher secretion of the two target cytokines in the ABMSCs group (P < 0.05).
Our data suggest that transplantation of ABMSCs promotes intestinal recovery after acute radioactive injury in Beagle dogs. The cytokines of IL-10 and MCP-1 might play an important role in this process.
Core Tip: Mesenchymal stem cell transplantation has a therapeutic role in intestinal radiation injury, but the exact mechanism is unknown. In the present study, we explored the therapeutic role of autologous bone marrow-derived mesenchymal stem cells (ABMSCs) and their possible mechanisms in a Beagle model of radioactive intestinal injury. Our experimental results suggest that ABMSC transplantation has a protective effect against acute radiation enteritis in Beagle dogs and that interleukin-10 and monocyte chemotactic protein-1 may play an important role in the treatment of ABMSCs.