Published online Sep 28, 2024. doi: 10.3748/wjg.v30.i36.4078
Revised: August 11, 2024
Accepted: September 6, 2024
Published online: September 28, 2024
Processing time: 102 Days and 21.2 Hours
This letter emphasizes the need to expand discussions on gut microbiome’s role in inflammatory bowel disease (IBD) and colorectal cancer (CRC) by including the often-overlooked non-bacterial components of the human gut flora. It highlights how viral, fungal and archaeal inhabitants of the gut respond towards gut dys-biosis and contribute to disease progression. Viruses such as bacteriophages target certain bacterial species and modulate the immune system. Other viruses found associated include Epstein-Barr virus, human papillomavirus, John Cunningham virus, cytomegalovirus, and human herpes simplex virus type 6. Fungi such as Candida albicans and Malassezia contribute by forming tissue-invasive filaments and producing inflammatory cytokines, respectively. Archaea, mainly metha-nogens are also found altering the microbial fermentation pathways. This corres-pondence, thus underscores the significance of considering the pathological and physiological mechanisms of the entire spectrum of the gut microbiota to develop effective therapeutic interventions for both IBD and CRC.
Core Tip: This letter to the editor intends to contribute to the conversation surrounding the role of gut microbiota in the progression of conditions such as inflammatory bowel disease and colorectal cancer. The letter emphasizes the importance of recognizing microbial components beyond bacteria that also play a significant role in the pathogenesis of these diseases, along with encouraging further studies in this area to better understand the role of viruses, fungi and archaea.