Published online Dec 28, 2022. doi: 10.3748/wjg.v28.i48.6950
Peer-review started: October 2, 2022
First decision: November 3, 2022
Revised: November 14, 2022
Accepted: December 13, 2022
Article in press: December 13, 2022
Published online: December 28, 2022
Processing time: 85 Days and 22.5 Hours
Colonic adenomatous polyposis of unknown etiology (CPUE) is an adenomatous polyposis phenotype that resembles Familial Adenomatous Polyposis (FAP) even though no germline pathogenic variant is identified.
We sought to better characterize the clinical features and outcomes in a cohort of CPUE patients.
This is a retrospective case series of patients 18 years old or older with aden-omatous oligopolyposis (between 10-100 adenomas) and negative genetic testing, identified through the Hereditary Gastrointestinal Cancer Database at Massachusetts General Hospital, a tertiary academic referral center. A retrospective chart review was performed with a focus on demographics, alcohol and tobacco use, medication use, familial malignancy and polyp burden, genetic testing information, endoscopic surveillance data including the corresponding histopathology, colonic and extracolonic malignancies, mortality events, and their etiology. Spearman correlation and Pearson Chi-square test (or Fisher's exact test) were used for continuous and categorical variables respectively.
CPUE patients were primarily male (69%) and presented for genetic counseling at 63.7 years. Only 2 patients (2.9%) reported a first-degree relative with polyposis. During an average surveillance period of 12.3 years, 0.5 colonoscopies per year were performed. Patients developed 2.3 new adenomas per year. 4 (5.7%) were diagnosed with colorectal cancer (CRC) at a mean age of 66 years, and 3 were diagnosed prior to the onset of oligopolyposis. 7 (10%) required colectomy due to advanced dysplasia or polyp burden. With respect to upper gastrointestinal manifestations, 1 patient had a gastric adenoma, but there were no cases of gastric or small bowel polyposis. During surveillance, 10 (14%) patients died at a mean age of 72, and none were due to CRC.
CPUE is distinct from familial adenomatous polyposis (FAP) syndrome and the use of FAP surveillance guidelines may result in unnecessarily frequent upper and lower endoscopies.
Core Tip: Colonic adenomatous polyposis of unknown etiology (CPUE) resembles familial adenomatous polyposis (FAP) syndrome, but no genetic alterations are identified. The optimal management of CPUE is uncertain. Patients with CPUE are typically older males that exhibit a low rate of new adenoma formation without upper gastrointestinal polyposis during long-term surveillance. 10% required colectomy for polyposis, and none died from colon cancer. The clinical behavior of CPUE is distinct from FAP, and the current application of FAP surveillance guidelines for CPUE may result in unnecessarily frequent upper and lower endoscopies.