Published online Sep 21, 2022. doi: 10.3748/wjg.v28.i35.5230
Peer-review started: January 27, 2022
First decision: April 10, 2022
Revised: April 13, 2022
Accepted: September 1, 2022
Article in press: September 1, 2022
Published online: September 21, 2022
Processing time: 231 Days and 9.2 Hours
It was reported that the urotensin II (U-II) level in inflammatory bowel disease (IBD) patients are significantly higher than in controls. To provide future guidance for the management of cardiovascular risk factors in IBD patients, the sample size of the current study appears to be limited, and more clinical samples to compare U-II levels in IBD patients and controls are needed. This will clarify the possible roles of inflammation factors and related signaling pathways (like EPK1/2, NF-κB and Rho/ROCK) in the pathophysiology of IBD. Therefore, large multicenter studies should be done to confirm the findings and underlying mech
Core Tip: An observational report showed that the level of urotensin II (U-II) in inflammatory bowel disease (IBD) patients was significantly increased compared with that in controls. The authors also reported that blood U-II level was positively correlated with high-sensitivity C-reactive protein, and severe endoscopic features of the disease. This study provides us with a new role of U-II in IBD, which warrants larger, multicenter clinical and basic studies to determine the mechanisms by which U-II triggers inflammatory responses and activates signaling pathways (EPK1/2, NF-κB and Rho/ROCK).