Published online Aug 21, 2019. doi: 10.3748/wjg.v25.i31.4437
Peer-review started: May 5, 2019
First decision: June 10, 2019
Revised: July 8, 2019
Accepted: July 19, 2019
Article in press: July19, 2019
Published online: August 21, 2019
Processing time: 111 Days and 1.4 Hours
Portal vein thrombosis (PVT) represents a well-known complication during the natural course of liver cirrhosis (LC), ranging from asymptomatic cases to life-threating conditions related to portal hypertension and hepatic decompensation. Portal flow stasis, complex acquired hypercoagulable disorders and exogenous factors leading to endothelial dysfunction have emerged as key factors for PVT development. However, PVT occurrence remains unpredictable and many issues regarding its natural history, prognostic significance and treatment are still elusive. In particular although spontaneous resolution or disease stability occur in most cases of PVT, factors predisposing to disease progression or recurrence after spontaneous recanalization are not clarified as yet. Moreover, PVT impact on LC outcome is still debated, as PVT may represent itself a consequence of liver fibrosis and hepatic dysfunction progression. Anticoagulation and transjugular intrahepatic portosystemic shunt are considered safe and effective in this setting and are recommended in selected cases, even if the safer therapeutic option and the optimal therapy duration are still unknown. Nevertheless, their impact on mortality rates should be addressed more extensively. In this review we present the most debated questions regarding PVT, whose answers should come from prospective cohort studies and large sample-size randomized trials.
Core tip: Portal vein thrombosis (PVT) represents a common and potential life-threating complication of liver cirrhosis. Anticoagulant therapy is advised in selected cases, in particular for liver transplant candidates. Despite the advanced knowledge in PVT pathogenesis and diagnosis, many issues regarding its natural history and prognostic outcome remain elusive. Likewise, the safer anticoagulant option, the potential role of direct oral anticoagulants and the optimal duration of therapy are still matter of debate. Given the clinical significance of this pathological entity, these cardinal issues should urgently be addressed in large prospective cohort studies and randomized trails.