Published online May 21, 2017. doi: 10.3748/wjg.v23.i19.3449
Peer-review started: December 9, 2016
First decision: January 19, 2017
Revised: January 23, 2017
Accepted: February 17, 2017
Article in press: February 17, 2017
Published online: May 21, 2017
Processing time: 162 Days and 22.4 Hours
To investigate the effects of heme oxygenase-1 (HO-1)-modified bone marrow mesenchymal stem cells (BMMSCs) on the microcirculation and energy metabolism of hepatic sinusoids following reduced-size liver transplantation (RLT) in a rat model.
BMMSCs were isolated and cultured in vitro using an adherent method, and then transduced with HO-1-bearing recombinant adenovirus to construct HO-1/BMMSCs. A rat acute rejection model following 50% RLT was established using a two-cuff technique. Recipients were divided into three groups based on the treatment received: normal saline (NS), BMMSCs and HO-1/BMMSCs. Liver function was examined at six time points. The levels of endothelin-1 (ET-1), endothelial nitric-oxide synthase (eNOS), inducible nitric-oxide synthase (iNOS), nitric oxide (NO), and hyaluronic acid (HA) were detected using an enzyme-linked immunosorbent assay. The portal vein pressure (PVP) was detected by Power Lab ML880. The expressions of ET-1, iNOS, eNOS, and von Willebrand factor (vWF) protein in the transplanted liver were detected using immunohistochemistry and Western blotting. ATPase in the transplanted liver was detected by chemical colorimetry, and the ultrastructural changes were observed under a transmission electron microscope.
HO-1/BMMSCs could alleviate the pathological changes and rejection activity index of the transplanted liver, and improve the liver function of rats following 50% RLT, with statistically significant differences compared with those of the NS group and BMMSCs group (P < 0.05). In term of the microcirculation of hepatic sinusoids: The PVP on POD7 decreased significantly in the HO-1/BMMSCs and BMMSCs groups compared with that of the NS group (P < 0.01); HO-1/BMMSCs could inhibit the expressions of ET-1 and iNOS, increase the expressions of eNOS and inhibit amounts of NO production, and maintain the equilibrium of ET-1/NO (P < 0.05); and HO-1/BMMSCs increased the expression of vWF in hepatic sinusoidal endothelial cells (SECs), and promoted the degradation of HA, compared with those of the NS group and BMMSCs group (P < 0.05). In term of the energy metabolism of the transplanted liver, HO-1/BMMSCs repaired the damaged mitochondria, and improved the activity of mitochondrial aspartate aminotransferase (ASTm) and ATPase, compared with the other two groups (P <0.05).
HO-1/BMMSCs can improve the microcirculation of hepatic sinusoids significantly, and recover the energy metabolism of damaged hepatocytes in rats following RLT, thus protecting the transplanted liver.
Core tip: Hepatic sinus is important in the liver microcirculation, which is the basis for transplanted liver regeneration. Transplanted liver grafts with disturbed microcirculation of the hepatic sinus may affect liver energy metabolism. We investigated the protective effects of heme oxygenase-1-modified bone marrow mesenchymal stem cells (HO-1/BMMSCs) on rat reduced-size liver transplantation in terms of the microcirculation and hepatic energy metabolism. HO-1/BMMSCs promoted the equilibrium of ET-1/NO, repaired damaged hepatic sinusoidal endothelial cells, and lowered the portal vein pressure in rats following reduced-size liver transplantation, which improved the microcirculation of hepatic sinusoids and ATPase activity, and recover the energy metabolism of damaged hepatocytes.