Published online Apr 14, 2017. doi: 10.3748/wjg.v23.i14.2566
Peer-review started: December 20, 2016
First decision: January 10, 2017
Revised: January 17, 2017
Accepted: March 15, 2017
Article in press: March 15, 2017
Published online: April 14, 2017
Processing time: 116 Days and 23.7 Hours
To elucidate the epidemiological characteristics and associated risk factors of perforated peptic ulcer (PPU).
We retrospectively reviewed medical records of patients who were diagnosed with benign PPU from 2010 through 2015 at 6 Hallym university-affiliated hospitals.
A total of 396 patients were identified with postoperative complication rate of 9.1% and mortality rate of 0.8%. Among 174 (43.9%) patients who were examined for Helicobacter pylori (H. pylori) infection, 78 (44.8%) patients were positive for H. pylori infection, 21 (12.1%) were on non-steroidal anti-inflammatory drugs (NSAIDs) therapy, and 80 (46%) patients were neither infected of H. pylori nor treated by any kinds of NSAIDs. Multivariate analysis indicated that older age (OR = 1.09, 95%CI: 1.04-1.16) and comorbidity (OR = 4.11, 95%CI: 1.03-16.48) were risk factors for NSAID-associated PPU compared with non-H. pylori, non-NSAID associated PPU and older age (OR = 1.04, 95%CI: 1.02-1.07) and alcohol consumption (OR = 2.08, 95%CI: 1.05-4.13) were risk factors for non-H. pylori, non-NSAID associated PPU compared with solely H. pylori positive PPU.
Elderly patients with comorbidities are associated with NSAIDs-associated PPU. Non-H. pylori, non-NSAID peptic ulcer is important etiology of PPU and alcohol consumption is associated risk factor.
Core tip: The incidence of complications of peptic ulcer has not been decreasing and only a few data is available about epidemiological characteristics and associated risk factors of perforated peptic ulcer. In a retrospective review of medical records from multicenter in Korea revealed that elderly patients with comorbidities were associated with non-steroidal anti-inflammatory drugs (NSAIDs)-associated peptic ulcer perforation and non-Helicobacter pylori (H. pylori), non-NSAID peptic ulcer is important etiology in the development of peptic ulcer perforation. In a multivariate logistic regression analysis, alcohol consumption was suspected to be associated risk factors for the development of non-H. pylori, non-NSAID peptic perforation.